1. Volume 106, Pages 26-32 (September 2017)
Is small intestinal bacterial overgrowth involved in the pathogenesis of functional dyspepsia? 
Georgios Tziatzios, Evangelos J. Giamarellos-Bourboulis, Ioannis S. Papanikolaou, Mark Pimentel, George D. Dimitriadis, Konstantinos Triantafyllou 
Medical Hypotheses 
Volume 106, Pages (September 2017)
DOI: /j.mehy
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2. Fig. 1
Mechanisms potentially contributing to functional dyspepsia pathogenesis; CNS: Central Nervous System.
Medical Hypotheses  , 26-32DOI: ( /j.mehy )
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3. Fig. 2
Schematic illustration of small bowel intestinal overgrowth development and functional symptoms generation by molecular mimicry mechanism that may apply to functional dyspepsia pathogenesis. Dotted lines indicate assumed steps in the process. ICC: interstitial cells of Cajal.
Medical Hypotheses  , 26-32DOI: ( /j.mehy )
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4. Fig. 3
Small bowel intestinal overgrowth (SIBO) – dysmotility interplay in functional dyspepsia (FD); ITT: Intestinal Transit Time.
Medical Hypotheses  , 26-32DOI: ( /j.mehy )
Copyright © 2017 Elsevier Ltd Terms and Conditions

5. Fig. 4
Small bowel intestinal overgrowth (SIBO) might be the “triggering” factor in a series of events causing increased intestinal permeability, intestinal dysmotility and subsequent functional symptom development.
Medical Hypotheses  , 26-32DOI: ( /j.mehy )
Copyright © 2017 Elsevier Ltd Terms and Conditions