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TWELVE-LEAD INTERPRETATION
By Leslie Hernandez, BS, NREMT-P, LP
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THE HEART One heart Two sides/Two arteries Three layers
Four chambers/valves
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BLOOD FLOW THROUGH THE HEART
From the Body Right Atrium To the Lungs Right Ventricle From the Lungs Left Atrium To the Body Left Ventricle
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THE ELECTRICAL CONDUCTION SYSTEM
SA Node Intranodal Pathways AV Junction AV Fibers Bundle of His Septum Bundle Branches Purkinje System
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The Electrical Conduction System
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THE ECG LEADS ECG Leads Bipolar Leads I, II, and II Unipolar
Leads aVR, aVL, and aVF Precordial V1, V2, V3, V4, V5, V6
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LEAD PLACEMENT
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THE ELECTROCARDIOGRAM
WAVES, INTERVAL, SEGMENTS AND COMPLEXES
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INTERVALS AND SEGMENTS
Time Intervals P–R Interval (PRI) or P–Q Interval (PQI) 120–200 ms QRS Interval 80–100 ms S–T Segment isoelectric Q–T Interval 360–440 ms
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P WAVE
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P-Waves and Atrial Enlargement
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THE PR INTERVAL ms
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THE QRS COMPLEX
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THE T WAVE
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How to Measure ST-Segment Deviation
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THE THREE I’S OF THE ACS S–T Segment Changes
Associated with Myocardial Infarctions Ischemia Injury Infarct
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Two Rules to Follow Changes >1 mm (0.1 mV) are significant If
They occur in two or more contiguous leads
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Patterns and Localization
Inferior – II, III, and aVF Septal – V1 and V2 Anterior – V3 and V4 Lateral – I, aVL, V5, and V6
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12-Lead ECG Variations in AMI and Angina
Baseline Ischemia—tall or inverted T wave (infarct), ST segment may be depressed (angina) Injury—elevated ST segment, T wave may invert Infarction (Acute)—abnormal Q wave, ST segment may be elevated and T wave may be inverted Infarction (Age Unknown)—abnormal Q wave, ST segment and T wave returned to normal
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Conditions that Mimic Injury
Pericarditis Diffuse or “global” ST-segment changes Left bundle branch blocks If possibly new, must treat as MI Left ventricular hypertrophy Causes ST-segment elevation Early repolarization Ventricular paced rhythms
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THE NORMAL TWELVE-LEAD
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ANTERIOR MI
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ANTEROLATERAL MI
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Inf. Wall Inj. Or Infarct with RCA
II, III, aVF, and V4R: Hypotension Supranodal and AV nodal blocks Atrial fibrillation / flutter PAC’s Significant NTG and MS hemodynamic hypersensitivity
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INFERIOR MI
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HEXAXIAL SYSTEM
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NORMAL AND ABNORMAL AXES
Normal QRS Axis Normal Left Axis (Physiologic) Abnormal Left Axis (Pathologic) Indeterminate Right Axis
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Axis Deviation Lead aVF QRS Lead I QRS Left Normal Axis Positive
Negative Lead aVF QRS Lead I QRS Right EXTREME RIGHT Left Normal Axis
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AXIS DEVIATION Left Axis Deviation Abnormal finding.
Often associated with hypertension, valvular heart disease, and other disease processes.
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Left Axis Deviation - Causes
Left ventricular enlargement Hypertension Aortic stenosis Ischemic heart disease Left bundle branch block and left anterior fascicular block
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RIGHT AXIS DEVIATION Right Axis Deviation Abnormal finding.
Often associated with COPD and pulmonary hypertension.
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Right Axis Deviation - Causes
Right ventricular enlargement COPD Pulmonary embolism Congenital heart diseases Other diseases causing pulmonary hypertension and cor pulmonale Right bundle branch block and left posterior fascicular block
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BUNDLE BRANCH BLOCKS Conduction Abnormalities Bundle Branch Blocks
Right Bundle Branch Block
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BUNDLE BRANCH BLOCKS Conduction Abnormalities The Turn-Signal Rule
QRS > 120 ms throughout the ECG. Look at the QRS in V1. Identify the J point. Draw a horizontal line. Triangle pointing up indicates RBBB. Triangle pointing down indicates LBBB.
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LEFT BUNDLE BRANCH BLOCK
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RIGHT BUNDLE BRANCH BLOCK
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CONDUCTION ABNORMALITIES
Hemiblocks Left Anterior Hemiblock
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CONDUCTION ABNORMALITIES
Hemiblocks Left Posterior Hemiblock
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CHAMBER ENLARGEMENT Chamber Enlargement Atrial Enlargement
Ventricular Hypertrophy Causes Right-sided enlargement and hypertrophy, usually secondary to long-term pulmonary disease. Left-sided enlargement and hypertrophy, usually secondary to long-term hypertension.
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RIGHT ATRIAL ENLARGMENT
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LEFT ATRIAL ENLARGEMENT
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RIGHT VENTRICULAR ENLARGEMENT
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LEFT VENTRICULAR ENLARGEMENT
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Time to Practice
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