1. Extensive cerebral infarction in the newborn due to incontinentia pigmenti 
Femke Maingay-de Groof, Maarten H. Lequin, Daniella W. Roofthooft, Arnold P. Oranje, Irenaeus F. de Coo, Levinus A. Bok, Peter J. van der Spek, Grazia M. Mancini, Paul P. Govaert 
European Journal of Paediatric Neurology 
Volume 12, Issue 4, Pages (July 2008)
DOI: /j.ejpn
Copyright © 2007 European Paediatric Neurology Society Terms and Conditions

2. Fig. 1
Patient 1: scheme of MR aspects on day 6; extensive signal intensity changes on T1- and T2- weighted sequences; involvement of cerebellum (arrow) depicted on a coronal FLAIR image; an image from the 3D T1 weighted gradient sequences with intravenous Gadolinium shows incomplete but extensive rupture of the blood brain barrier in several arterial fields; the circle of Willis is intact (except for non-filling of the A1 part on the left, arrow) on the 3D phase contrast (PC) sequence performed 3 days later. Definite abnormalities within the large arteries may not have been observed due to insufficient resolution of MRA images.
European Journal of Paediatric Neurology  , DOI: ( /j.ejpn )
Copyright © 2007 European Paediatric Neurology Society Terms and Conditions

3. Fig. 2
Patient 2: neonatal brain US scans in coronal and parasagittal (right) views: the arrows point to hyperechoic streaks along striatal arteries, similar to the aspect of arteritis in congenital infections like CMV.
European Journal of Paediatric Neurology  , DOI: ( /j.ejpn )
Copyright © 2007 European Paediatric Neurology Society Terms and Conditions

4. Fig. 3
Patient 2: sagittal T2-weighted MRI at 4 years: extensive atrophy of cerebellar vermis in the left panel; stripes of hypersignal from gliosis (arrows) in white matter of the centrum semi-ovale in a parasagittal section between insula and thalamus (right panel). Very thin corpus callosum probably due to atrophy.
European Journal of Paediatric Neurology  , DOI: ( /j.ejpn )
Copyright © 2007 European Paediatric Neurology Society Terms and Conditions