1. Cytogenetic and molecular events in adenoma and well-differentiated thyroid follicular- cell neoplasia 
Paola Caria, Roberta Vanni 
Cancer Genetics and Cytogenetics 
Volume 203, Issue 1, Pages (November 2010)
DOI: /j.cancergencyto
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2. Fig. 1 Tumors originating from thyroid hormone–producing cells.
Cancer Genetics and Cytogenetics  , 21-29DOI: ( /j.cancergencyto )
Copyright © 2010 Elsevier Inc. Terms and Conditions

3. Fig. 2
Common cytogenetic numerical changes in adenoma. A representative G-banded karyotype with 54 chromosomes is shown.
Cancer Genetics and Cytogenetics  , 21-29DOI: ( /j.cancergencyto )
Copyright © 2010 Elsevier Inc. Terms and Conditions

4. Fig. 3
Common cytogenetic structural changes in benign thyroid proliferation. A partial karyotype showing a t(5;19)(q13;q13) is shown.
Cancer Genetics and Cytogenetics  , 21-29DOI: ( /j.cancergencyto )
Copyright © 2010 Elsevier Inc. Terms and Conditions

5. Fig. 4
Schematic representation of chromosome abnormalities leading to fusion genes in papillary thyroid carcinoma (a–c), adenoma, and follicular carcinoma (d).
Cancer Genetics and Cytogenetics  , 21-29DOI: ( /j.cancergencyto )
Copyright © 2010 Elsevier Inc. Terms and Conditions

6. Fig. 5
Schematic representation of bacterial artificial chromosome clones flanking RET (a, left) and PPARγ (b, left) used to reveal gene disruption via the fluorescence in situ hybridization break-apart probe strategy (a and b, right). Arrows point to the break-apart signals.
Cancer Genetics and Cytogenetics  , 21-29DOI: ( /j.cancergencyto )
Copyright © 2010 Elsevier Inc. Terms and Conditions