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Lecture Notes Chapter 1: Introduction to Patient Assessment.

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1 Lecture Notes Chapter 1: Introduction to Patient Assessment

2 Objectives  Define the role of the respiratory therapist in
patient assessment List the most common symptoms associated with cardiopulmonary disease and the common causes of each List common physical procedures performed to evaluate patients with cardiopulmonary disease and the implications of abnormalities Explain the laboratory tests done to evaluate patients with diseases of the chest and common causes of abnormalities

3 Introduction  Patient assessment is a vital part of patient care in
any setting Emergency department Intensive care unit (ICU) Home-care setting  Clinicians best at patient assessment are those who are rigorous in their daily patient care

4 Medical History and Interview
 Gathers important information about what is wrong with the patient  Allows rapport to be established  Requires clinician’s knowledge about diseases  Ask appropriate questions

5 Medical History and Interview  Basic questions 
When did the problem start? Have you ever had this problem before? How severe is the problem? Has the severity changed since onset? What aggravates or relieves the problem? What region of the body is affected? Answers to these questions will help determine a differential diagnosis, what further tests may be needed, and what initial therapy may be appropriate.

6 Medical History and Interview 
Symptoms: Shortness of Breath (Dyspnea) Common symptom in cardiopulmonary disease Usually worsens with exertion and improves with rest. Dyspnea at rest indicates poor cardiopulmonary reserve May worsen with position  Orthopnea: reclining (supine) – heart failure  Platypnea: upright – post lung surgery Dyspnea occurring with change in position is abnormal. Any pathologic lung change that increases the work of breathing usually will lead to dyspnea, especially during exertion.

7 Medical History and Interview  Symptoms: Cough 
One of the most common symptoms of cardiopulmonary disease Cough receptors of large airways are stimulated: Mechanical(occurs when foreign bodies touch the airway chemical (irritating gas), inflammatory, thermal (cold air). Common causes wall.), Acute respiratory infections Asthma Gastroesophageal reflux Post-nasal drip Identify severity, frequency, associated sputum production weak cough may indicate thoracic or abdominal pain or neuromuscular disease.

8 Medical History and Interview  Symptoms: Sputum Production 
Mucus is normally produced in small amounts in the airways, defense against invasion by germs. It is moved to the larynx by cilia. It is not normally noticed by healthy people. Sign of disease of the airways or lungs Most common cause: cigarette smoking  Other causes: inflammation – asthma/pneumonia Quality Mucoid = airway disease without infection (clear and thick) Fetid = infection by anaerobes (foul Smelling) Purulent = bacterial infection (contains pus, green or yellow) Copious = bronchiectasis ( large amounts)

9 Medical History and Interview  Symptoms: Chest Pain 
Pleuritic chest pain Is made worse by breathing, coughing, sneezing, moving chest wall Sharp in nature Associated with pleuresy, pneumothorax, embolism, pneumonia pulmonary Nonpleuritic chest pain Located centrally in the chest Usually not affected by respiratory efforts It may radiate – shoulder, arm, jaw, back Dull pressure Associated with ischemic heart disease

10 Medical History and Interview  Symptoms: Hemoptysis 
Spitting up of blood from tracheobronchial Etiology tree Pneumonia Trauma Bronchitis/bronchiectasis Bronchogenic carcinoma Tuberculosis Pulmonary embolism Massive hemoptysis = 200–600 mL/day

11 Medical History and Interview 
Symptoms: Fever increase in body temperature caused disease by Most often a manifestation of infection Increases O2 consumption and CO2 production Common respiratory problems associated with fever Viral infections Bacterial bronchitis, pneumonia Fungal infections Tuberculosis Immunocompromised patients may not generate fever despite severe infection The degree of fever depends on the severity of the infection the patient’s ability to respond to the infection. and

12 Medical History and Interview  Symptoms: Wheeze 
Airway wall vibration between partially open and closed position as air passes speed Common in through at high Asthma Congestive heart failure Bronchitis Usually associated with dyspnea Wheezing may be associated with cough, sputum, and dyspnea when airway disease is present.

13 Physical Examination  To confirm or rule out suspected illness from
interview  To identify abnormalities not even related to the chief complaint  RTs = to evaluate response to therapy

14 Physical Examination: Vital Signs (VS) and Sensorium
 VS provide an index of patient’s acute condition  Heart rate  Tachycardia = > 100 beats/min  Anxiety, hypoxemia, heart failure, fever, bronchodilators  Bradycardia = < 60 beats/min  Hypothermia, SA node damage, vagal stimulation

15 Physical Examination: (VS) and Sensorium Vital Signs
 Respiratory rate  Tachypnea = > 20 breaths/min  Pulmonary disease  Pain, anxiety, fever, exertion  Bradypnea = < 10 breaths/min Vital Signs (not common)  Hypothermia and CNS disorders

16 Physical Examination: (VS) and Sensorium Vital Signs
 Blood pressure (120/80) Vital Signs Hypotension = < 90/60 mm Hg  Heart failure, vasodilatation, hypovolemia  Puts patient at risk for shock Hypertension = > 140/90 mm Hg  Risk factor for heart failure and stroke  Cause often unknown

17 Physical Examination: (VS) and Sensorium Vital Signs
 Body temperature Vital Signs 37°C +/- 0.5°C (98.6°F +/- 1°F) Fever = infections Hypothermia  Head injury that damage the hypothalamus, drowning, medications Body temperature affects O2 consumption near

18 Physical Examination: Vital (VS) and Sensorium Signs
Sensorium assessment Patient’s level of consciousness (LOC) Reflects Adequacy of cerebral blood flow Adequacy of brain oxygenation Net effect of acid–base balance Electrolyte balance Nutritional status Integrity of other organ systems The patient who is alert and oriented as to time, place, 3”. Confusion and person is said to be “oriented times patient is disoriented. Disoriented patient may indicate respiratory failure

19 Physical Examination: Vital (VS) and Sensorium Signs
 Pulse oximetry “Fifth vital sign” Signs Intermittent or continuous monitoring oxygenation status of Reads amount of light absorption and determines degree of oxygenation Falsely high if HbCO present Falsely low if poor perfusion

20 Physical Examination: Chest Inspection
 Breathing pattern it can provide important clues regarding the underlying lung pathology Prolonged expiratory phase  Intrathoracic airway obstruction asthma, bronchitis. Prolonged inspiratory phase  Extrathoracic airway obstruction airway (epiglottitis) Rapid and shallow breathing  Restrictive lung disease Abdominal paradox narrowed upper  Diaphragmatic fatigue common in patients with acute exacerbations of COPD.

21 Physical Examination: Chest Inspection
 Degree of symmetrical chest expansion  Deformities Scoliosis – lateral curvature of spine Kyphosis – A-P curvature of the spine Kyphoscoliosis – both Barrel chest – COPD Pectus excavatum – sternal depression Pectus carinatum – sternal protrusion

22

23 Physical Examination: Chest Palpation
Tactile fremitus patient is asked to repeat the phrase “1- 2-3” or “99” while the examiner palpates the comparing side to side. chest wall Increased in presence of lung densities  Pneumonia Decreased when lung is less dense  Emphysema Decreased if pleural space is occupied  Pleural effusion, pneumothorax Presence of crepitation on the skin  Subcutaneous emphysema

24 Physical Examination: Chest Percussion
 Tapping (chest percussion) to detect changes in lung density  Normal resonant sound  Decreased resonance (dull)  Increased lung density or pleural effusion  Increased resonance Decreased lung density Pneumothorax Pneumonia

25 Physical Examination: Auscultation Chest
 Information about  Patency of the airway  Condition of lung parenchyma  Normal breath sounds Chest Tracheal heard directly over the trachea Bronchovesicular heard around the sternum on the anterior chest wall *** between the scapulae on the posterior chest wall. Vesicular over the areas of the chest wall

26 Physical Examination: Auscultation Chest
 Abnormal breath sounds Chest Bronchial breath sounds  Pneumonia, atelectasis, fibrosis Diminished breath sounds Loss of lung parenchyma = emphysema Diseases of the chest or pleural space Shallow breathing

27 Physical Examination: Auscultation Chest
 Continuous adventitious breath Chest sounds Wheezing (intrathoracic) Musical quality Expiratory Polyphonic = high-pitched = asthma/CHF Monophonic = low-pitched = single airway obstruction Stridor (extrathoracic) Usually inspiratory

28 Physical Examination: Chest Auscultation
 Discontinuous adventitious breath sounds Crackles Sudden opening of small airways Fine crackles Small airways Late inspiration Movement of excessive secretions with breathing Coarse crackles Large airways Inspiration and expiration

29 Physical Examination: Examination of Other Parts of the Body
 Neck  Jugular Venous Distention degree angle  Right ventricular failure  Precordium (JVD) evaluated at 45 Ventricular heave  Ventricular hypertrophy Normal heart sounds “lub-dub” (S1, S2) Gallops (S3, S4) in heart failure Murmurs valves stenosis

30 Physical Examination: Examination of Other Parts of the Body
 Extremities  Digital clubbing COPD  Peripheral cyanosis  Abdomen  Distension  Hepatomegaly  Mouth ~ circulatory failure  Oral cyanosis ~ respiratory failure  Pursed-lip breathing COPD to maintain patency airways during exhalation. ofdistal

31 Interpretation of Lab Data: and Fluid Tests Blood
 Complete blood count (CBC) Blood White blood cells (WBCs)  Leukocytosis vs. leukopenia Red blood cells (RBCs)  Polycythemia vs. anemia Hemoglobin (Hb)

32 Interpretation of Lab Data: Blood and Fluid Tests
 Serum Electrolytes to evaluate the general health condition Sodium (Na+) Potassium (K+) Chloride (Cl-) Bicarbonate (HCO3-)

33 Interpretation of and Fluid Tests Lab Data: Blood
 Microbiology section  Gram stain  Culture  Cardiac enzymes Lab Data: Blood Creatin phosphokynase Troponin (CPK) (CPK) and troponin enzymes are usually elevated within hours of a heart attack.

34 Interpretation of Blood Gases Lab Data: Arterial  Oxygenation 
 PaO2: 90–100 mm Hg Lab Data: Arterial Acid–base pH: 7.35–7.45 Acidemia vs. alkalemia  Hypoxemia: mm Hg  Ventilation < 60 Respiratory metabolic vs.  PaCO2: 35–45 mm Hg  Hypercapnia vs. hypocapnia

35 Interpretation of Lab Data: Pulmonary Function Testing
Lung volumes  VC the amount of gas fully exhaled after a maximum inhalation  Restrictive (due to low lung volumes) vs. obstructive (narrowed airways) Airway patency  FEV1forced expiratory volume in1 second  FEF 25%–75% the middle half of the forced vital capacity. These measures of flow help determine whether obstructive lung disease is present.

36 Interpretation of Radiographs: Chest Radiograph
 Identifies changes as disease changes lung density  Identifies proper position of ETT: 3–5 cm above carina Chest tube Nasogastric tube  Types of densities identified Bone Water Fat Air Radiopaque vs. radiolucid

37 Interpretation of Radiographs: Chest Radiograph
 Radiopaque = increased density  Pneumonia  Tumors  Radiolucid = decreased density  Emphysema  Pneumothorax  Cardiac size  Cardiomegaly

38 Interpretation of Clinical Chest Radiograph Lab Data:
 Computerized tomography Lab Data: More detail images Cross-sectional view Best for evaluation of tumors

39 Interpretation of Clinical Lab Data
 Electrocardiography  Recording electrical activity of the  Assessment of  Cardiac rate and rhythm  Status of the heart muscle  Waves Lab Data heart P QRS T


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