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Subarachnoid Haemorrhage
Dr. Jonathan Yang Neurosurgery
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SAH Sudden onset of severe headache SAH until proven otherwise !!!
5-10% of all non-traumatic intracranial haemorrhage 15/ population
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Aetiology Most common one for Adults Rupture of a berry aneurysm 70%
Paeds much less common, and most a/w AVM AVM Adult 10% Tumor, bleeding disorders, blood dyscrasia 5% Idiopathic 15%
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Presenting features Sudden onset of a severe headache not previously experienced by the patient. Deterioration of conscious state Meningism Focal neurological signs Intracerebral haemorrhage, local pressure effects, cerebral vasospasm
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Grades Hunt and Hess grading system 1 - 5 WFNS grading system 1 -5
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Investigations CT Best initial investigations LP, Xanthochromia
Confirm the dx in 85% Provide A/W pathology LP, Xanthochromia Correct handling Cerebral Angiography, CTA, DSA
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Cerebral Aneurysm Most common cause of SAH in adult
Max in 4th and 5th decades of life Arises at the branch points of two vessels 85% anterior half of the circle of Willis 15% posterior half of the circle of Willis Multiple aneurysm occur in 15% of cases
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Pathogenesis Saccular, berry or congenital aneurysm Fusiform aneurysm
Arises at the junction of vessels Congenital deficiency in the muscle coat Discontinuity of the underlying internal elastic membrane Fusiform aneurysm HTN
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Aneurysm (Related facts)
Seven fold in first degree relatives 2-5% results in SAH (Lifetime) Hereditary syndromes Ehlers-Danlos Syndrome Coarctation of the Aorta Polycystic kidney disease
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Management of ruptured cerebral aneurysm
Three factors Severity of the initial haemorrhage 30% cases death or deeply comatosed Rebleeding of the aneurysm Occurs in about 50% of patients within 6 weeks 25% of patients within 2weeks of the initial haemorrhage. Half of the patients die Prevention VS Risks Cerebral Vasospasm
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Cerebral vasospasm Angiographic vasospasm occurs in about 50% of patients following SAH 25% with serious outcome Direct correlation between the amount of blood noted in the basal cisterns. Does not usually occur until 2-3 days after the initial bleed. And rarely delayed after 14days Mx Hypertensive, Hypervolaemia, Ca antagonist
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Mx of Aneurysm Surgery Endovascular obliteration of the aneurysm
Occlusion of the neck Reinforcement of the sac Proximal ligation Endovascular obliteration of the aneurysm The access Configuration
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AVM Most common cause of SAH in children Haemorrhage Epilepsy
Progressive neurological deficit Surgery Size Radiosurgical techniques Less than 3cm
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