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Published byEleanore King Modified over 6 years ago
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دکترامیر هوشنگ واحدی متخصص طب فیزیکی و توانبخشی قسمت 1
Abnormal gait دکترامیر هوشنگ واحدی متخصص طب فیزیکی و توانبخشی قسمت 1
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pathologic gait is an inefficient mode of locomotion that usually requires considerably more energy than a normal gait. Patients may adopt many kinds of abnormal movements to minimize their energy usage
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Effects of pathological gait
Increased energy expenditure, body will use compensatory mechanisms to maintain balance Position the center of gravity over the base of support Use other body movements to allow clearance of foot and forward progression of body
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Etiology of pathological gait
1. Muscle weakness 2. Contractures or deformities 3. Spasticity 4. Joint instability 5. Skeletal instability 6. Shortening (LLD) 7. Impaired motor control (from the brain) 8. Sensory loss 9. Pain
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Classification of Gait Abnormalities
Anatomical Classification Functional Classification Pathological Classification
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Anatomical Classification
Region Sagittal Frontal Transverse Foot Toe flexion Pes planus/supinatus In/out-toeing Ankle Equinus, Drop foot Excessive dorsiflexion Varus/Valgus Tibia Tibial Torsion Knee Stiff knee Recurvatum Abnormal loading Femur Femoral Anteversion Hip Forward trunk flexion Tendelenberg sign Pelvis Anterior/posterior tilt Rotation Spine Lordosis/kyphosis Duchenne sign Scoliosis Arms Abnormal swing
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Functional Classification
Velocity Stride Length Cadence Stance Duration Step Length Step Time Step Width Antalgic Low Short Fast Long Wide Unstable Compensated Normal Uncompensated Apropulsive Reduced
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Pathological Classification
Pathology Predominantly Symmetrical Predominantly Assymetrical Upper motoneuron (UMN) Diplegia Quadriplegia Hemiplegia Brain Stem Ataxic Lower motoneuron (LMN) Spina Bifida Femoral nerve palsy Sensory Neuropathy Basal Ganglia Athetoid Cortical Apraxia Orthopedic Anteversion Tibial torsion Congenital Dislocation (CDH) Amputee Trans-tibial Trans-femoral
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Abnormal gait in relation of anatomical abnormalities
Cortex : circumduction gait, Apraxic gait Spinal cord: scissoring gait Peripheral nerves: motor fiber foot drop,high step gait sensory fiber : sensory ataxia NMJ: waddling gait Muscle: waddling gait Basal ganglia: short step gait, Shuffling gait Cerebellum: cerebellar ataxia,Tandem gait Joint and bone: Limping gait
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Antalgic Gait Gait pattern in which stance phase on affected side is shortened Corresponding increase in stance on unaffected side avoidance of bearing full weight on the affected leg limitation of range of movement
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Antalgic gait Conditions associated
Trauma Osteoarthritis Pelvic girdle pain Coxalgia tarsal tunnel syndrome
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Apraxic gait difficulty initiating a step freezing
feet almost stuck to floor turn hesitation shuffling gait hypokinesia muscular rigidity grasp reflexes possible resting tremor, dementia or urinary incontinence
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Gait cadence disturbances
During all phase of gait cycle Stride length and time spent on the affected limb differ from those in the contra lateral limb or from a normal gait Pain of falling: During stance phase patient takes a short, rapid step with the affected leg Correspondingly, slower step with sound leg
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Cadence Disturbances Neurological disease: Ataxia, cerebrovascular disorders, proprioceptive deficits and spasticity, impaired balance Unequal length: Skeletal shortening, contracture, tendon shrinkage, locked joints or ankle joint stops introduce asymmetry into the gait
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Unilatera=Trendelenberg gait Bilateral = waddling gait
Lateral Trunk bending Unilatera=Trendelenberg gait Bilateral = waddling gait Patient leans towards the affected stance leg as weight is transferred
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Trendelenburg Gait Weak hip abductors cannot do this and compensate by tilting trunk to weight bearing side, shifting CoG closer to hip joint During stance phase in coronal plane Pelvis tilts laterally towards the over stand leg
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Common causes: 1.Painful hip 2.Hip abductor weakness
3.Leg-length discrepancy 4.Abnormal hip joint 5.Hip Abductor contracture 6.Hip dislocation
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