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History Taking in Respiratory Diseases
Suleiman Momany, MBBS, FCCP
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Why to Take History? 70% of medical problems can be diagnosed by proper history taking 20% can be diagnosed after physical exam
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Patient Centered NOT Clinician Centered
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5 – Step Model of Interviewing
Set the stage for interview Elicit the chief complaint Open the history of present illness Continue the patient centered history of present illness Transition to clinician centered process
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A 29-year old woman presented to pulmonary clinic complaining of dry cough for the last three months. She noticed that her cough is worse at night and after exposure to cold air and household chemicals. She also has associated wheezes and chest tightness. She feels short of breath when going upstairs. Has seasonal running nose and sneezing and watery eyes. She visited ER twice for this complaint. She smokes ½ pack per day and works as hair dresser. Had childhood ? Asthma Family history of allergic rhinitis She is currently taking occasional antihistamine and received nebulized treatment in the ER.
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Basic Requirements for History Taking
Satisfactory approach to the patient Give patient adequate time to tell and express himself/herself Competent interrogation and skillful communication
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History Taking in Respiratory Diseases
Major symptoms Past history Family history Occupational history Social history
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Major Symptoms Upper respiratory tract Lower respiratory tract
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Upper respiratory tract symptoms
Nasal obstruction Nasal discharge Sneezing Epistaxis Sore throat Hoarseness Stridor Cough
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Lower respiratory tract symptoms
Cough Sputum Hemoptysis Chest pain Dyspnea Wheeze
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Cough The 5th most common symptom seen in outpatient clinics
An explosive expiration that clears the tracheobronchial tree from secretions and foreign materials Intrathoracic pressure may reach 300 mmHg and expiratory velocity 800 km/h
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Cough Onset Duration Diurnal variation Dry or productive
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Causes of Cough
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Sputum Amount Character Viscosity Taste and odor Serous Mucoid
Purulent Rusty Viscosity Taste and odor
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Types of Sputum
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Hemoptysis Amount: massive Type: frank, streaks, clots Duration
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Causes of Hemoptysis
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Chest pain Central Lateral Trachea Heart Vessels Esophagus Pleuritic
Herpes zoster Root compression
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Causes of Non-Central Chest Pain
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Causes of Central Chest Pain
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Dyspnea Unpleasant and unexpected awareness of breathing
Factors contributing to dyspnea Increased work of breathing Increased ventilatory drive Impaired respiratory muscle function
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Increased work of breathing
Airflow limitation Decreased compliance Restricted expansion Increased pulmonary ventilation Increased dead space Metabolic acidosis Severe hypoxia Hysterical Weakness of respiratory muscles Poliomyelitis Myasthenia gravis Spinal cord injury
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Causes of Dyspnea
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Causes of Dyspnea
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Dyspnea: onset, duration and progression
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NYHA Severity Grading of Dyspnea
Grade I (minimal): dyspnea on running or on doing more than ordinary effort Grade II : dyspnea on ordinary effort Grade III: dyspnea on doing less than ordinary effort Grade IV: dyspnea at rest
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MRC Dyspnea Scale
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Variants of Dyspnea Orthopnea PND: Paroxysmal nocturnal dyspnea
Platypnea
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Wheeze Wheezes are continuous high pitched (400Hz) musical sounds produced by oscillations of airway walls. The oscillations begins when the airflow velocity reaches a critical value called flutter velocity. Wheezes always accompanied by air flow limitation Invariably louder during expiration and may be confined to expiration Rhonchi are low pitched sounds (200Hz) Stridor is a high-pitched wheezing sound resulting from turbulent airflow in upper airways
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Causes of wheezes
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Past history Childhood illnesses; measles and whooping cough
Tuberculosis Pneumonia Chest trauma Chest (or other surgeries) Previous x-rays Recent anaesthesia or loss of consciousness Previous Hospitalization; Ward Vs ICU Admission Previous Mechanical Ventilation
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Medication History Follow up of disease severity ( SABA Calendar)
Response to treatment Proper Device use Side effects Drug Induced Lung Disease
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Drug Induced Lung Disease
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Family history Atopy Cystic fibrosis Tuberculosis
Chronic obstructive lung disease Cancer
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Occupational and Environmental history
Chemicals Organic dust Animal proteins Non-organic dust
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The Spectrum of Occupational Lung Disease
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Hypersensitivity pneumonitis / (Extrinsic Allergic Alveolitis)
A spectrum of granulomatous, interstitial, and alveolar-filling lung diseases Result from repeated inhalation of and sensitization to a wide variety of organic dusts
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Pneumoconioses Group of interstitial lung diseases caused by the inhalation of certain dusts and the lung tissue’s reaction to the dust Primary pneumoconiosis are asbestosis, silicosis, and coal dust Other forms aluminum, antimony, barium, graphite, iron, kaolin, mica, talc, among other dusts. There is also a form called mixed-dust pneumoconiosis. Typically many years Some cases – silicosis, particularly – rapidly progressive forms can occur after only short periods of intense exposure
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Asbestos exposure Asbestosis = parenchymal disease interstitial fibrosis Pleural Calcification Malignant mesothelioma Lung cancer: all types
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Lung Cancer Occupational Exposure Several occupational carcinogens
Environmental exposures Outdoor particulate matter Residential radon exposure (attributable risk 7% in US) Environmental tobacco smoke Enviromental exposure to occupational carcinogens low dietary intake; other factors Sources: Samet J. J Natl Cancer Inst 1989 and Steenland K et al. Am J Ind Med 1996
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Occupational lung cancer occupations
Aluminium production Coal gasification Coke production Hematite mining Iron and steel founders Painting Rubber production Source :IARC
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Occupational lung cancer agents
Arsenic Asbestos BCME Beryllium Cadmium Chromium 6 Silica dust Nickel Ionizing radiation Occupational exposure to strong inorganic acids Sulfur mustard Polycyclic aromatic hydrocarbons Soot Coal tar pitch Diesel exhaust
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Occupational History
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Social history Smoking (Pack-Year) Alcohol
Keeping pets (birds or animals)
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