1. Effectiveness of sugammadex for cerebral ischemia/reperfusion injury
Sule Ozbilgin, Osman Yılmaz, Bekir Ugur Ergur, Volkan Hancı, Seda Ozbal, Serhan Yurtlu, Sakize Ferim Gunenc, Bahar Kuvaki, Burcu Ataseven Kucuk, Ali Rıza Sisman 
The Kaohsiung Journal of Medical Sciences 
Volume 32, Issue 6, Pages (June 2016)
DOI: /j.kjms
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2. Figure 1
Effects of sugammadex treatment on neuronal density, crezyl-violet staining (A1–4), TUNEL (B1–4), and active caspase-3 immunoreactivity (C1–4) in the CA1 region of the hippocampus. Neuronal density was significantly less in the IR group (A2). TUNEL and active caspase-3-positive cells enhanced in the IR group (B2–C2). Sugammadex (100 mg/kg) treatment significantly reduced the number of apoptotic neurons (B4–C4). Arrows indicate TUNEL-positive and active caspase-3 immune-positive cells. IR = ischemia–reperfusion; TUNEL = terminal deoxynucleotidyl transferase dUTP nick end labeling.
The Kaohsiung Journal of Medical Sciences  , DOI: ( /j.kjms )
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3. Figure 2
Effects of sugammadex treatment on neuronal density, crezyl-violet staining (A1–4), TUNEL (B1–4) and active caspase-3 immunoreactivity (C1–4) in the parietal cortex. The neuronal density is significantly less in the IR group (A2). TUNEL and active caspase-3-positive cells enhanced in the IR group (B2–C2). Sugammadex (100 mg/kg) treatment significantly reduced the number of apoptotic neurons (B4–C4). IR = ischemia–reperfusion; TUNEL = terminal deoxynucleotidyl transferase dUTP nick end labeling.
The Kaohsiung Journal of Medical Sciences  , DOI: ( /j.kjms )
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