1. From: Male Prevalence of Acquired Color Vision Defects in Asymptomatic Carriers of Leber’s Hereditary Optic Neuropathy
Invest. Ophthalmol. Vis. Sci ;48(5): doi: /iovs
Figure Legend:
(A) Left: stimulus from the CCT, showing spatial and luminance noise in the background and in the Landolt C chromatic target. The subject’s task was to indicate the position of the gap, which is changed randomly among right, left, up, and down from one trial to the next. Right: section of the CIE 1976 chromaticity diagram showing the coordinates for the stimuli used in the CCT Trivector procedure. The CRT gamut for the monitor we used is represented by the colored area, which is delimited by the phosphor chromaticities: red phosphor (R) u′ = 0.416; v′ = 0.522; green phosphor (G) u′ = 0.117; v′ = 0.559; blue phosphor (B) u′ = 0.159; v′ = in u′v′ chromaticity coordinates. The chromaticity of the target (Landolt C) varied along the protan (P), deutan (D), and tritan (T) confusion axes. The background chromaticity was fixed and it was located at the point where the axes converge. Thresholds were determined along each of the confusion axes. (B) Average and SE of the thresholds of asymptomatic LHON carriers (n = 46) and control subjects (n = 74) in the Trivector test of the CCT for the protan, deutan, and tritan axes. Statistical significance was observed between control subjects and carriers for all three confusion axes. However, threshold elevation was larger for the protan and deutan axes. (C) Protan, deutan, and tritan thresholds from the LHON carriers and control subjects, presented in (B) were normalized to the respective average value in the control group. (▵) Carrier thresholds; (○) control thresholds; horizontal bars: tolerance limits.
Date of download: 10/26/2017
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