1. Chapter 67: Non-Parathyroid Hypercalcemia
Mara J. Horwitz, Steven P. Hodak, and Andrew F. Stewart

2. Table 1. Differential Diagnosis of Hypercalcemia
From the Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 7th Edition.
Table 1. Differential Diagnosis of Hypercalcemia
© 2008 American Society for Bone and Mineral Research

3. From the Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 7th Edition.
Figure 1
Figure 1 Comparison of bone histology in a patient with HPT (A), HHM (B), and LOH caused by leukemia (C). In HPT, HHM, and LOH, osteoclastic activity is accelerated (large, thick arrows), although it is higher in HHM than in HPT. In HPT, osteoblastic activity (thin arrows) and osteoid are increased, but both are markedly decreased in HHM and LOH. This uncoupling of formation from resorption in HHM and LOH plays the major rule in causing hypercalcemia. (Reprinted with permission from Stewart AF, Vignery A, Silvergate A, Ravin ND, LiVolsi V, Broadus AE, Baron R 1982 Quantitative bone histomorphometry in humoral hypercalcemia of malignancy: uncoupling of bone cell activity. J Clin Endocrinol Metab 55:219–227.) Copyright 1982, The Endocrine Society
Figure 1 Comparison of bone histology in a patient with HPT (A), HHM (B), and LOH caused by leukemia (C). In HPT, HHM, and LOH, osteoclastic activity is accelerated (large, thick arrows), although it is higher in HHM than in HPT. In HPT, osteoblastic activity (thin arrows) and osteoid are increased, but both are markedly decreased in HHM and LOH. This uncoupling of formation from resorption in HHM and LOH plays the major rule in causing hypercalcemia. (Reprinted with permission from Stewart AF, Vignery A, Silvergate A, Ravin ND, LiVolsi V, Broadus AE, Baron R 1982 Quantitative bone histomorphometry in humoral hypercalcemia of malignancy: uncoupling of bone cell activity. J Clin Endocrinol Metab 55:219–227.) Copyright 1982, The Endocrine Society