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Lets talk about Ba examination

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Presentation on theme: "Lets talk about Ba examination"— Presentation transcript:

1 Lets talk about Ba examination

2 Radiology of G.I.T Radiological investigations 1- Contrast examination Barium study Ba. Swallow, Ba. Meal, Ba. Follow through & Ba enema 2-Endoscopic Ultrasound . 3- CT & MRI. 4-Nuclear medicine (FDG-PET): flurodeoxyglucose positron emission tomography

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4 THE OESOPHAGUS

5 THE OESOPHAGUS Barium Swallow: 1. Conventional 2
THE OESOPHAGUS Barium Swallow: 1. Conventional 2. Double contrast (DC) 3. Flouroscopy + spot films

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8 Technique 1.Patient will need to be NPO after midnight before the exam
2.The patient will have to swallow a contrast agent: Barium or Gastrograffin May also swallow sodium bicarbonate for double contrast barium swallow 3.X-ray tech will have the patient perform various maneuvers so that the barium can coat the GI tract

9 Indications Dysphagia Hematemesis Abdominal pain Odynaphagia
Unexplained weight loss

10 The Normal Anatomy:. Long tubular structure. Length 25-30 cm
The Normal Anatomy: *Long tubular structure * Length cm * Start at level of C5 ( Crico-pharengeal) * End – Cardiac sphincter * Three portions Cervical Thoracic Abdominal * Normal Mucosal pattern (DC) thin regular longitudinal Parallel Numbers of lines ( 4-5 )

11 Areas of normal Narrowings - body of the cervical vertebra - AA - LT atrium - diaphragmatic hiatus

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13 On the Hypo pharyngeal part Common
structures that we can visualize are: Epiglottis Post cricoid impression lateral pharyngeal pouches Crico pharyngeal muscle impression

14 LEFT: Lateral view: Epiglottis (red arrow).
Post cricoid impression (yellow arrows). Crico pharyngeous impression (white arrow). RIGHT: AP-view: Small lateral pharyngeal pouches (arrows)

15 Mucosa The esophageal wall is composed of: Musculature
Inner circular layer Outer longitudinal layer: Upper 1/3 striated muscle Middle 1/3 striated and smooth muscle Lower 1/3 smooth muscle No serosa

16 Esophagus mucosa: normal thin, parallel, uniform mucosal folds 3-4 in no.in double contrast examination

17 Esophageal peristalsis
Normal: Primary contraction: Propels bolus through the esophagus Secondary contraction: Follows primary contraction and propels any remaining bolus from thoracic esophagus

18 Abnormal contraction :
Tertiary contractions, Diffuse esophageal spasm crock screw o. ))Nutcracker esophagus Decreased peristalsis resulting from achalasia, scleroderma, dermatomyositis, polymyositis, esophagitis, and secondary to many other diseases

19 tertiary contractions

20 Diffuse esophageal spasm
Diffuse esophageal spasm produces intermittent contractions of the mid and distal esophageal smooth muscle, associated with chest symptoms

21 Congenital Anomalies 1- Artesia with or with out tracheo-oesophageal fistula (TEF). 2- Congenital Short oesophagus. 3- Congenital Duplication ( Neuro-enteric cyst ) ATRESIA: - Complete blockage of the lumen . - The diagnosis is suggested after birth by in ability of infant to feed or by choking during swallowing . - The blocked segment is mostly seen at level of thoracic inlet

22 Types of Fistula

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26 Acquired Lesions DYSPHAGIA difficulty in swallowing causes
1- Carcinoma ( Malignant stricture). 2-Benign Stricture (Corrosive ). 3-Achalasia Cardia. 4-forgien body 5-osophagitis .

27 Malignant stricture CA esophagus is the cause for the malignant stricture The most common types of esophageal carcinoma are squamous cell carcinoma and adenocarcinoma . esophagography is unique among esophageal studies for assessing both morphology and motility. Barium esophagography remains the study of choice for characterization of esophageal strictures. Esophageal carcinoma may demonstrate a variety of appearances on barium esophagrams.

28 Annular Carcinoma Narrowing : 1-Constant. 2-Irrigular . 3-Variable length. 4- Shouldering sign. 5-Fistula (double tract). 6-Soft tissue shadow of the mass

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32 Computed Tomography Contrast-enhanced CT plays an important role in the 1.staging of esophageal carcinoma. to 2.determining the extent of the local tumor; 3.invasion of mediastinal structures; 4.involvement of supra clavicular, mediastinal, or upper abdominal lymph nodes 5. Assessment of the distant metastases

33 examination should extend from the thoracic inlet through the liver Routine oral contrast material such as (gastrographine) or a negative intra luminal contrast medium, such as water. +/ - IV contrast injection CT essential in the Dx & staging of the CA

34 CT finding of esophageal malignancy
1.Eccentric or circumferential wall thickening is greater than 5 mm. 2.Peri-esophageal soft tissue and fat stranding may be demonstrated. 3.A dilated fluid- and debris-filled esophageal lumen is proximal to an obstructing lesion. 4.Aortic invasion . 5.Osophageal CA is often metastatic at the time of presentation ( look for the LN & distal metastasis ) .

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37 Barrett's esophagus is a metaplastic disorder in which specialized columnar epithelium replaces healthy squamous epithelium. Barrett's metaplasia is the most common cause or precursor of esophageal carcinoma. The rate of esophageal adenocarcinoma is increasing in the Western world, and it is associated with a poor prognosis, mainly because individuals present with late-stage disease..

38 Benign Stricture Causes : Peptic esophagitis Corrosive Traumatic
Ba. swallow : 1-Constant narrowing. 2- Long length (lower third). 3-Smooth and regular. 4-Mild proximal dilatation. 5-No shouldering sign. 6-Smooth tapering ( funnel shape).

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40 Inflammation and Infection Gastroesophageal reflux (GERD) is the most common cause of esophagitis.

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42 Infectious esophagitis
Candida esophagitis in patient with an infectious esophagitis due to candida , the barium shows numerous fine erosions & plaques causing shaggy outline of the osophagus due to Candida albicans in immunocompromised patient.

43 middle year old female with a past medical history significant for HIV/AIDS comes in with complaint of loosing their weight over the past 2 weeks with pain & difficulty on swallowing …. Also feels like food is getting stuck in her throat What is your diagnosis ??????????

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