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Brain Function and Head Injuries

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Presentation on theme: "Brain Function and Head Injuries"— Presentation transcript:

1 Brain Function and Head Injuries

2 Bony Anatomy

3 Bony Anatomy

4 Bony Anatomy The Cephalic Region has 28 bones:
The “skull” is called the cranium Its bones (flat bones) are “sutured” together forming Synarthrodial (fibrous – immovable – joints) The mandible is the only moveable bone Temporomandibular joint (TMJ) Combination hinge and gliding (Synovial) joint supported by 3 main ligaments (Temporomandibular, Stylomandibular & Sphenomandibular) Masseter – muscle prime mover for chewing (3 other muscles also help) & talking (along w/tongue) Mastication = chewing

5 Brain Anatomy Frontal lobe

6 Brain Facts The brain: Weighs about 3 lbs:
75% of the weight is water 85% by weight is cerebrum Fattest organ in the body Doesn’t feel pain – there are no pain receptors in the brain Requires 20% of body’s oxygen Requires 15-20% of the blood supply – there are 100,000 miles of blood vessels in the brain Has billions of cells (neurons)- (brain cells do not reproduce)

7 More Brain Facts Transmits information through neurons
100 billion – 1 trillion neurons 1,000 to 10,000 synapses for each neuron Process information at speeds of up to 268 mph. Neurons do not reproduce – but new connections can be created. Information is sent electrically or chemically (neurotransmitters) Axon transmits info across synapse Dendrite receives information

8 Even More Brain Facts Neuroplasticity capacity of neurons and neural networks in the brain to change their connections and behavior in response to new information, sensory stimulation, development, damage, or dysfunction. Neurotransmitters – chemical agents that assist information transfer such as: Endorphins: Associated with emotions and pain perception. Dopamine: Associated with thought and pleasurable feelings.

9 3 Main Brain Areas Cerebrum: 2 hemispheres - The right hemisphere of the brain controls the left side of the body (and vice versa) Frontal Lobe: reasoning & problem solving, voluntary muscle movement, emotions & eye movement Parietal Lobe: Sensation, perception, movement Occipital Lobe: Vision Temporal Lobe: Hearing, memory & speech Cerebellum: equilibrium, coordination, posture, muscle actions & some reflexes Brain Stem: controls automatic functions (i.e. breathing)

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11 Cerebrospinal Fluid It is a clear, colorless fluid
It’s primary functions are: Protect the brain from impacts – keeps tissue buoyant absorbing shock Bathe the brain in chemicals for proper functioning (delivering nutrients/removing waste products) Help maintain regular pressure between cranium and spinal column In severe head injury cases may drain from nose or ear.

12 The Scalp Contains a large # of blood vessels muscles and hair.
Lacerations of the scalp bleed profusely A blow to the head may cause bleeding under the skin – a hematoma – or visible lump Can significantly reduce the force of impact to the cranium.

13 Preventing Head Injuries
Use of proper safety equipment including helmets, face masks and mouth pieces. Adhering to safety rules. Teaching and using proper technique. Using safety videos to demonstrate dangers of the sport. Using common sense.

14 Head Injury Mechanisms
The most common cause is direct impact. The most susceptible area is the temporal region (thinnest bone). Contrecoup injuries are also possible. Whiplash like event with head hitting ground/surface Rotation of the head (after initial impact) can also cause injury. Nerve receptors overload can cause unconsciousness.

15 Skull Fractures Types of fractures include:
Depressed – pushes part of skull toward the brain. Bleeding under skin or from laceration Linear – fracture straight across the skull No displacement but blood vessels under the skull are torn. Compound – part of the skull sticking out through the scalp – profuse bleeding. Penetrating – object goes through scalp, skull & often the brain.

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17 Concussions Temporary impairment ( a type of TBI – traumatic brain injury) of brain function caused by impact or rotation. Symptoms include: Confused, dazed or unconscious Nausea, dizziness, loss of balance, headache Vomiting, difficulty speaking, disorientation Tinnitus – ringing in the ears Amnesia – loss of memory Battle Sign – discoloration behind the ear

18 Concussions (cont’d) Severe symptoms – Can lead to paralysis or death
Unconsciousness Also include: Rapid eye movement (fluttering eyes) Unequally dilated pupils (pupil on injury side will be enlarged if head injury is serious. Coma (although the athlete will be able to hear) Increased blood pressure, decreased pulse rate, shock Can lead to paralysis or death

19 Concussions ATC must check for other injuries that may be caused during the impact – especially a neck or spine injury. In the cases of moderate or severe concussions the athlete should be back boarded (head & neck immobilized) The athlete should be monitored for 24 hours as some symptoms maybe delayed Must rule out intracranial hematoma (severe bleeding within the brain) a potentially fatal condition. An athlete who has had a concussion is 4 times more likely to suffer another one.

20 Recent concussion statistics

21 Comparison of Cantu Concussion Guidelines (1986 & 2001)
Grade 1 (mild)   1986 Cantu No loss of consciousness (LOC) Posttraumatic amnesia (PTA) lasting less than 30 minutes 2001 Cantu (Evidence Based) No LOC PTA lasting less than 30 minutes OR Post concussion signs or symptoms lasting less than 30 minutes

22 Comparison of Cantu Concussion Guidelines (1986 & 2001)
2nd Degree (moderate) 1986 Cantu LOC lasting less than 5 minutes OR PTA lasting longer than 30 minutes but less than 24 hours 2001 Cantu (Evidence Based) LOC lasting less than 1 minute AND PTA lasting longer than 30 minutes but less than 24 hours OR Post concussion signs or symptoms lasting longer than 30 minutes but less than 24 hours

23 Comparison of Cantu Concussion Guidelines (1986 & 2001)
3rd Degree (severe) 1986 Cantu LOC lasting longer than 5 minutes OR PTA for more than 24 hours 2001 Cantu (Evidence Based) LOC lasting longer than 1 minute OR PTA for more than 24 hours OR Post concussion signs or symptoms lasting longer than 7 days

24 ImPACT Testing and Return to Activity
Baseline test done prior to participation results good for 2 calendar years. Incident occurs – initial evaluation of concussion Athlete’s parents are notified Athlete is sent to physician or ER Recovery – Student’s next school day completes symptom check list Low score - student takes ImPACT test. High score – student must wait additional 24 hours Failed ImPACT test student must wait 3 days before retaking (each failure) Academic accommodations and/or referral to concussion specialist for severe persistence of symptoms.

25 ImPACT Testing and Return to Activity
Recovery (cont’d) Athlete must pass ImPACT test Be symptom free for 24 hours after attending normal school day (w/o accommodations) and watching practice 5 day return to play progression Walking, stationary bike, jog/run, non-contact activities Length of workouts increased over progression Any re-occurrence of symptoms stops the progression 6th day – athlete returns to play

26 Related conditions Post concussion syndrome – Second-impact syndrome –
a persistence of symptoms Usually does not last more than 1-2 weeks Can become a long term problem with repeated concussions Second-impact syndrome – When an athlete receives more than one concussion in a short period of time.

27 Related conditions CTE – (Chronic Traumatic Encephalopathy)
Degenerative condition of the brain in athletes with repetitive brain trauma from both concussions and sub concussive events Build up of Tau protein in the brain causes dysfunction First identified in boxing as “punch drunk” syndrome in the 1920’s Symptoms are not present for years or decades afterwards

28 CTE cont’d The symptoms of CTE include: Memory loss Confusion
Impaired judgment Impulse control problems Aggression Depression Anxiety suicidality, parkinsonism & progressive dementia.

29 CTE cont’d Not known: Why some end up with CTE and others don’t
How many impacts does it take to trigger CTE changes No current way to diagnose CTE (including through CAT scan or MRI)

30 Concussion interview & video


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