1. Core I97L mutation in conjunction with P79Q is associated with persistent low HBV DNA and HBs antigen clearance in patients with chronic hepatitis B 
T. Honda, M. Ishigami, Y. Ishizu, T. Kuzuya, K. Hayashi, T. Ishikawa, Y. Murakami, M. Iwadate, H. Umeyama, H. Toyoda, T. Kumada, Y. Katano, H. Goto, Y. Hirooka 
Clinical Microbiology and Infection 
Volume 23, Issue 6, Pages 407.e1-407.e7 (June 2017)
DOI: /j.cmi
Copyright © 2016 European Society of Clinical Microbiology and Infectious Diseases Terms and Conditions

2. Fig. 1
(a) Cumulative incidence of low hepatitis B virus (HBV) DNA and normal alanine aminotransferase (ALT) in patients with the I97L mutation and wild-type I97. (b) Cumulative incidence of clearance of hepatitis B surface antigen (HBsAg) in patients with the I97L mutation and wild-type I97. (c) Cumulative incidence of low HBV DNA and normal ALT in patients with I97L+P79Q mutation, I97L + P79, and I97 + P79. (d) Cumulative incidence of clearance of HBsAg in patients with I97L + P79Q, I97L + P79, and I97 + P79. The number of patients at risk is shown below the graphs (a–d).
Clinical Microbiology and Infection  , 407.e1-407.e7DOI: ( /j.cmi )
Copyright © 2016 European Society of Clinical Microbiology and Infectious Diseases Terms and Conditions