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Defects in DBA: more than meets the eye
by Lydie Da Costa Blood Volume 122(6): August 8, 2013 ©2013 by American Society of Hematology
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The defect in erythroid progenitors from RPL5+/R23X iPSs generated from DBA affected fibroblasts (A), where most of the cells are myeloid cells, and the rescue by the wild RPL5 transgene, where most of the cells are erythroid progenitors (B). The defect in erythroid progenitors from RPL5+/R23X iPSs generated from DBA affected fibroblasts (A), where most of the cells are myeloid cells, and the rescue by the wild RPL5 transgene, where most of the cells are erythroid progenitors (B). See the complete Figure 7 in the article by Garçon et al that begins on page 912. Lydie Da Costa Blood 2013;122: ©2013 by American Society of Hematology
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