1. Erythropoietin’s Beta Common Receptor Mediates Neuroprotection in Spinal Cord Neurons 
Lisa S. Foley, MD, David A. Fullerton, MD, Joshua Mares, BA, Mitchell Sungelo, BA, Michael J. Weyant, MD, Joseph C. Cleveland, MD, T. Brett Reece, MD 
The Annals of Thoracic Surgery 
Volume 104, Issue 6, Pages (December 2017)
DOI: /j.athoracsur
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2. Fig 1
Primary spinal cord neurons in culture. Primary spinal cord neurons are identified by a network of projections when cultured to maturity.
The Annals of Thoracic Surgery  , DOI: ( /j.athoracsur )
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3. Fig 2
Neuron viability after transduction. LV2 resulted in the least cell loss after transduction experiments. (LV = lentiviral vector.)
The Annals of Thoracic Surgery  , DOI: ( /j.athoracsur )
Copyright © 2017 The Society of Thoracic Surgeons Terms and Conditions

4. Fig 3
Beta common receptor expression after transduction with knockdown virus. LV1, LV2, and LV4 successfully suppressed beta common receptor expression. Based on these findings along with the previous viability findings, LV2 was chosen as the knockdown virus used for all further experiments. (LV = lentiviral vector.)
The Annals of Thoracic Surgery  , DOI: ( /j.athoracsur )
Copyright © 2017 The Society of Thoracic Surgeons Terms and Conditions

5. Fig 4
Spinal cord neuron viability after oxygen-glucose deprivation (OGD). Erythropoietin (EPO) significantly preserved neuronal viability after OGD treatment. EPO-mediated attenuation of neuronal injury was lost in beta common receptor (βcR) knockdown cells compared with nonsense control cells. (∗Clinical significance, p < 0.05.)
The Annals of Thoracic Surgery  , DOI: ( /j.athoracsur )
Copyright © 2017 The Society of Thoracic Surgeons Terms and Conditions