Download presentation
Presentation is loading. Please wait.
Published byRodrigo Carlos Abreu Modified over 6 years ago
1
Cardiometabolic Risk in Type 2 Diabetes: What Can the PPAR-g Agonists Teach Us?
2
UKPDS 35: Impact of Increasing A1c on Cardiovascular Disease in T2DM
3
Insulin Resistance Is Associated With or Directly Responsible for Factors Causing Atherothrombosis
4
UKPDS 35: Effect of A1c on MI and Microvascular Events
5
UKPDS 80: 10-Year Follow-up Study of Intensive Glycemic Control in T2DM
6
Incidence of CV Events in Subjects With T2DM vs the Nondiabetic CAD Population
7
PPAR-g Agonists: Lipoprotein Effects
8
PROactive: Time to Secondary CV Composite End Point
9
FDA Statistical Review and Evaluation Meta-analysis of Pioglitazone
10
CV Events With PPAR-g Agonists
11
Genes Uniquely Regulated by the TZDs
12
Pioglitazone Induces IkBa Protein Expression In Vivo in a PPAR-a-Dependent Manner
13
The Concept of Dual PPAR-a/g Agonists
14
Effect of Muraglitazar on Composite CV Endpoints
15
Effect of Tesaglitazar on Serum Creatinine and Glomerular Filtration
16
SYNCHRONY: Effect of Aleglitazar vs Pioglitazone on A1c
17
SYNCHRONY: Effect of Aleglitazar vs Pioglitazone on LDL-C and HDL-C
18
ALECARDIO: Aleglitazar in Patients With T2DM and ACS
19
Program Summary
Similar presentations
© 2025 SlidePlayer.com. Inc.
All rights reserved.