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Facial Nerve Paralysis
Dr. Vishal Sharma
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Gabriel Fallopius ( )
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Anatomy of Facial Nerve
Motor root: 7000 axons Sensory root (Nervus intermedius / Wrisberg): 3000 axons. Joins motor root at fundus of I.A.C. Motor: predominantly to facial muscles Secretomotor: lacrimal, submandibular, sublingual Taste: anterior 2/3rd of tongue Sensory: Post-aural / concha / ext. auditory canal
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Course of facial nerve
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Parts of facial nerve Intracranial: within cerebello-pontine angle
Intra-temporal Meatal segment Labyrinthine segment Tympanic segment Mastoid segment Extra-cranial Extra-parotid Intra-parotid (terminal)
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Segments of Facial Nerve
1. Supranuclear: Fibers in cerebral cortex to brain stem 2. Brain stem: Motor nucleus of facial nerve (pons) 3. Intra-cranial (12 mm): Brain stem to entry into IAC 4. Meatal (10 mm): Within Internal Auditory Canal 5. Labyrinthine (4 mm): Fundus of I.A.C. to Geniculate gangl. 6. Tympanic (11 mm): Geniculate ganglion to pyramid 7. Mastoid (13 mm): Pyramid to stylomastoid foramen 8. Extra-temporal (15 mm): S.M. foramen to pes anserinus
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Primary branches of facial nerve
Intra-temporal: greater superficial petrosal, stapedius, chorda tympani Extra-parotid: post-auricular, stylohyoid, posterior belly of digastric Intra-parotid: temporal, zygomatic, buccal, marginal mandibular, descending cervical
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Intra-cranial branches
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Extra-cranial branches
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Communicating branches to:
Meatal: vestibulo-cochlear Tympanic: lesser petrosal otic ganglion Mastoid: auricular branch of vagus Extra-parotid: glossopharyngeal, auriculotemporal, vagus, greater auricular, lesser occipital Terminal: branches of trigeminal
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Surgical landmarks
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Cochleariform process: small bony protuberance
(from which tensor tympani muscle turns 900 to insert into malleus) lies 1 mm inferior to geniculate ganglion at anterior end of tympanic segment. Cog: bony ridge hanging from tegmen tympani lies 1 mm above & posterior to cochleariform process. Incus short process: 2 mm below lies external genu Lateral Semicircular Canal: 2 mm Antero-Infero-Medial lies external genu Oval window: 1 mm above lies external genu
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Inferior edge of Posterior S. C. C
Inferior edge of Posterior S.C.C.: 2 mm anterior & lateral lies mastoid segment of facial nerve Tympano-mastoid suture in posterior canal wall: 5-8 mm medial lies mastoid segment of facial nerve Digastric ridge in mastoid tip: leads antero-medially to mastoid segment of facial nerve Groove between mastoid & bony E.A.C. meatus: bisected by facial nerve Tragal pointer: 1 cm antero-infero-medial is facial nv Root of styloid process: lateral lies facial nerve Superior border of posterior belly of digastric: superior & parallel lies facial nerve
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Surgical landmarks
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Lesions of Facial Nerve
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Lesion Manifestation Supranuclear C/L hemiplegia, ed jaw jerk
Nuclear (pons) I/L 6th, 7th palsy + C/L hemiplegia In C.P. Angle I/L 5th, 7th, 8th palsy Supra-geniculate ed lacrimation, hyperacusis, loss of taste Supra-stapedial Hyperacusis, loss of taste Supra-chordal Loss of taste Infra-chordal Facial asymmetry only
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Upper Motor Neuron Palsy Lower Motor Neuron Palsy
Features Upper Motor Neuron Palsy Lower Motor Neuron Palsy Forehead wrinkling B/L present Same side absent Eye closure Naso-labial fold Opposite side absent Drooping of angle of mouth Opposite side Same side
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Etiology of Facial Nerve Palsy
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1. Idiopathic (55%): Bell’s palsy,
Melkersson Rosenthal syndrome 2. Temporal bone trauma (25%): Road traffic accident 3. Infection (10%): C.S.O.M., Herpes Zoster oticus Malignant otitis externa 4. Neoplasm (5%): Parotid tumors, Acoustic Neuroma, Glomus tumors, Malignancy of ear 5. Congenital (4%): Moebius syndrome 6. Iatrogenic (rare): Mastoidectomy, Parotid surgery 7. Metabolic (rare): Diabetes mellitus, Hypertension
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Sunderland’s Classification (1951)
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Cross section of nerve
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Partial block of axoplasm Injury to endoneurium or myelin sheath
Grade Name Characteristics I Neuropraxia Partial block of axoplasm II Axonotemesis Injury to axon III Neurotemesis Injury to endoneurium or myelin sheath IV Partial transection Injury to perineurium V Complete transection Injury to epineurium
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House Brackmann Classification (1 year post-injury)
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Grade Characteristics I II III IV V VI
Description Characteristics I Normal Normal facial function II Mild dysfunction Slight weakness seen only on close inspection III Moderate dysfunction Obvious asymmetry; complete eye closure IV Moderately severe dysfunction Obvious asymmetry; incomplete eye closure V Severe dysfunction Only minimal motion seen; asymmetry at rest VI Total paralysis No movement
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House Brackmann grading
Sunderland Grading EEMG response Recovery begins in House Brackmann grading I Normal 1-4 wks II 25 % of normal 1-2 mth III < 10 % of normal 2-4 mth III or IV IV No response 4-18 mth V Never VI
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Diagnosis Topo-diagnostic Tests Electrical Tests
Magnetic stimulation of intra-cranial facial nerve CT scan temporal bone: for progressive palsy MRI brain Surgical exploration
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Topo-diagnostic tests
Audiometry: cochlear nerve function Vestibulometry: vestibular function Schirmer’s test: Greater Superficial Petrosal Nerve Stapedial reflex test: Nerve to stapedius Electrogustometry: Chorda tympani Submandibular salivary flow: Chorda tympani Examination for terminal facial nerve branches
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Schirmer’s Test Unilateral wetness ed by >30% of total amount of
both eyes after 5 minutes = Schirmer test positive lesion at or proximal to geniculate ganglion
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Stapedial Reflex
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Electrogustometry Measures minimum amount of current required to excite sensation of taste
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Muscles supplied by terminal branches
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Electrical tests
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Nerve Excitability Test
Stimulating electrode used over terminal branches of facial nerve Minimum current intensity required to produce minimal muscle movement is calculated Normal side compared to paralyzed side Difference > 3.5 mAmp = unfavorable prognosis
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Maximal stimulation test
Stimulating electrode used over terminal branches of facial nerve Minimum current intensity required to produce maximal muscle movement is calculated Normal side compared to paralyzed side Difference > 3.5 mAmp = unfavorable prognosis
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Electro-neuronography
Terminal branch of facial nerve stimulated & action potential recorded in appropriate muscle Paralyzed side compared to normal side (which is taken as 100%) Response > 10% = % chance of recovery Response < 10% = 25 % chance of recovery
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Electro-neuronography
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Electro-neuronography
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Electro-neuronography
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Records spontaneous activity of facial muscles
Electromyography Records spontaneous activity of facial muscles
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Electromyography Responses
Normal Polyphasic Fibrillation Electrical Silence
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Response Interpretation
Normal Motor Unit Action Potentials: Incomplete transection of facial nerve Poly-phasic Motor Unit Action Potentials: Re-innervation of facial muscles Fibrillation potentials: Denervation of muscles (2-3 weeks after trauma) Electrical silence: Atrophy / absence of muscle
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Bell’s Palsy Acute onset, idiopathic, unilateral, self-limiting, non-progressive, peripheral facial nerve palsy 85% start recovering within 3 weeks Etiology: 1. Viral: Herpes simplex, Herpes Zoster 2. Ischemia of facial nerve: exposure to cold, emotional stress, nerve compression 3. Hereditary Autoimmune
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Sir Charles Bell
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Clinical Features Loss of forehead wrinkles Inability to close eyes
Wide palpebral fissure Epiphora Loss of naso-labial fold Drooping of angle of mouth Dribbling of food while chewing on affected side
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Medical treatment Prednisolone (1mg/kg in 2 doses): for 2 - 3 weeks
Acyclovir: mg 5 times per day X 7days Eye care: Voluntary 2 / min. Ciplox eye drops 2 hourly & ointment H.S. Eye cover at night. Physiotherapy: moist heat + facial massage + facial muscle exercise Electrical stimulation of facial nerve & muscle Facial nerve decompression: Controversial
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Moebius syndrome
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Melkersson Rosenthal Syndrome
Recurrent alternating facial palsy Fissured tongue Facio-labial edema Familial history
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Melkersson Rosenthal Syndrome
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Surgical Treatment for Facial Nerve Injury
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A. Facial nerve decompression: till meatal foramen
B. Neurorrhaphy (Nerve repair) 1. Direct end to end anastomosis 2. Interposition Cable grafting: sural, greater auricular C. Nerve Transposition: hypoglossal-facial D. Muscle Transposition: temporalis, masseter E. Micro-neuro-vascular muscle flaps F. Static Procedures: eyelid implant, fascial sling
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Treatment Protocol Up to 3 weeks: Nerve decompression or Nerve repair
3 weeks – 2 year: Nerve Repair or Nerve Transposition > 2 year with fibrillation in Electromyography: Nerve Repair or Nerve Transposition > 2 yr with electrical silence in Electromyography: Muscle transposition / Eyelid implant / Fascial sling
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Facial Nerve Decompression
Cortical mastoidectomy done Facial nerve canal bone thinned in barber pole fashion with diamond burr. Drilling done: Posteriorly at mastoid segment, Laterally at external genu & Inferiorly at tympanic segment Avoids injury to chorda tympani & lateral S.C.C. Labyrinthine segment decompressed by middle cranial fossa approach
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Barber Pole
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Direct repair & Cable Grafting
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Nerves used for cable grafting
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Nerve Transposition
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Nerve Transposition
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Temporalis muscle transposition
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Masseter muscle transposition
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Gold Weight Eyelid Implant
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Complications of facial nerve injury
1. Incomplete recovery 2. Exposure keratitis 3. Facial tics & spasms 4. Faulty regeneration of facial nerve a. Synkinesis: Mass movement of facial muscles b. Crocodile tear syndrome: gustatory lacrimation Salivary to lacrimal gland cross over c. Frey’s syndrome: gustatory sweating Secreto-motor to sympathetic cross over
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Thank You
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