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Figure 4 Relation of neuropsychological deficits and intrathecal immune cell subsets in GABAB receptor antibody–associated limbic encephalitis Relation.

Published byCordelia Alexina Sharp Modified over 6 years ago

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Presentation on theme: "Figure 4 Relation of neuropsychological deficits and intrathecal immune cell subsets in GABAB receptor antibody–associated limbic encephalitis Relation."— Presentation transcript:

1 Figure 4 Relation of neuropsychological deficits and intrathecal immune cell subsets in GABAB receptor antibody–associated limbic encephalitis Relation of neuropsychological deficits and intrathecal immune cell subsets in GABAB receptor antibody–associated limbic encephalitis Relation of neuropsychological deficits to the absolute numbers of CD138+ CD19+ plasma cells (A), activated HLADR+ CD4+ T cells (B), and activated HLADR+ CD8+ T cells (C) and within the CSF in 3 patients with GABAB receptor antibody–associated limbic encephalitis. The overall neuropsychological deficits are quantified as the total neuropsychological percentile rank sum, and the verbal memory retrieval performance is quantified as the respective percentile rank sum (see table e-2). For details, refer to the main text. GABAB = γ-aminobutyric acid B; Pt. = patient. Kristin S. Golombeck et al. Neurol Neuroimmunol Neuroinflamm 2016;3:e232 © 2016 American Academy of Neurology

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Figure 1 Treg percentage and suppressive function increased during each round of Treg infusions Treg percentage and suppressive function increased during.
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Figure Immune checkpoint inhibitor–induced encephalitis before and after treatment with natalizumab Immune checkpoint inhibitor–induced encephalitis before.
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