1. Gating Immunity and Death at the Nuclear Pore Complex
Mary Dasso, Beatriz M.A. Fontoura 
Cell 
Volume 166, Issue 6, Pages (September 2016)
DOI: /j.cell
Copyright © 2016 Elsevier Inc. Terms and Conditions

2. Figure 1
Nuclear Pore Rearrangement upon NB-LRR Signaling Induces Effector-Triggered Immunity and Programmed Cell Death
The activation of nucleotide-binding leucine-rich repeat receptor proteins (NB-LRR) results in disruption of CPR5 oligomerization, which causes the release of cyclin-dependent kinase inhibitors (CKIs) sequestered by CPR5 and rearrangement of trafficking through the nuclear pore complex. Together, these effects cause immune/stress signaling cargos to enter the nucleus to trigger transcription of genes that encode proteins that function in effector-triggered immunity (ETI) and programmed cell death (PCD).
Cell  , DOI: ( /j.cell )
Copyright © 2016 Elsevier Inc. Terms and Conditions