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Pancreaticobiliary Maljunction
Terumi Kamisawa, Kensuke Takuma, Hajime Anjiki, Naoto Egawa, Masanao Kurata, Goro Honda, Kouji Tsuruta, Tsuneo Sasaki Clinical Gastroenterology and Hepatology Volume 7, Issue 11, Pages S84-S88 (November 2009) DOI: /j.cgh Copyright © 2009 AGA Institute Terms and Conditions
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Figure 1 ERCP of a patient with PBM without biliary dilatation.
Clinical Gastroenterology and Hepatology 2009 7, S84-S88DOI: ( /j.cgh ) Copyright © 2009 AGA Institute Terms and Conditions
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Figure 2 Mechanism of biliary carcinogenesis in PBM.
Clinical Gastroenterology and Hepatology 2009 7, S84-S88DOI: ( /j.cgh ) Copyright © 2009 AGA Institute Terms and Conditions
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Figure 3 (A) MRCP of a patient with CCC showing markedly dilated extrahepatic bile duct with a long common channel. (B) Reflux of contrast medium into the main pancreatic duct was detected in multidetector-row CT with drip infusion cholangiography in the same patient. Clinical Gastroenterology and Hepatology 2009 7, S84-S88DOI: ( /j.cgh ) Copyright © 2009 AGA Institute Terms and Conditions
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Figure 4 ERCP of a patient with HCPBD and a common channel of 9 mm in length (A). The communication between pancreatic and bile ducts was eliminated by sphincter contraction (B). Clinical Gastroenterology and Hepatology 2009 7, S84-S88DOI: ( /j.cgh ) Copyright © 2009 AGA Institute Terms and Conditions
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