1. Experience with repair of congenital heart defects using adjunctive endovascular devices 
John G. Coles, MDa, Ilya Yemets, MDa (by invitation), Hani K. Najm, MDa (by invitation), Jeanne M. Lukanich, MDa (by invitation), Jean Perron, MDa (by invitation), Greg J. Wilson, MDc (by invitation), Marlene Rabinovitch, MDb (by invitation), David G. Nykanen, MDb (by invitation), Lee N. Benson, MDb (by invitation), Ivan M. Rebeyka, MDa (by invitation), George A. Trusler, MDa, Robert M. Freedom, MDb, William G. Williams, MDa 
The Journal of Thoracic and Cardiovascular Surgery 
Volume 110, Issue 5, Pages (November 1995)
DOI: /S (95)
Copyright © 1995 Mosby, Inc. Terms and Conditions

2. Fig. 1
A, Preoperative angiogram in neonate with pulmonary atresia and VSD showing ductal insertion onto origin of left pulmonary artery producing potential for pulmonary arterial confluence stenosis. B, Severe, bilateral proximal pulmonary artery stenosis that occurred four months after insertion of endovascular stents across origin of both branch pulmonary arteries and inflation of stents to 5mm diameter.
The Journal of Thoracic and Cardiovascular Surgery  , DOI: ( /S (95) )
Copyright © 1995 Mosby, Inc. Terms and Conditions

3. Fig. 2
Section taken through right upper lobe pulmonary vein immediately upstream of site of endovascular stent implantation.Severe intimal hyperplasia is present, attributable to smooth muscle cell proliferation and extensive extracellular matrix material, consisting of predominantly glycosaminoglycans, collagen, and disorganized elastic fiber production (Movat stain; original magnification x 20).
The Journal of Thoracic and Cardiovascular Surgery  , DOI: ( /S (95) )
Copyright © 1995 Mosby, Inc. Terms and Conditions

4. Fig. 3
Autopsy specimen illustrating satisfactory left ventricular position of 17 mm USCI clamshell device. Marked mitral annular and left ventricular hypoplasia were present.
The Journal of Thoracic and Cardiovascular Surgery  , DOI: ( /S (95) )
Copyright © 1995 Mosby, Inc. Terms and Conditions