1. Primary biliary cirrhosis: A 2010 update
Raoul Poupon 
Journal of Hepatology 
Volume 52, Issue 5, Pages (May 2010)
DOI: /j.jhep
Copyright © 2010 European Association for the Study of the Liver Terms and Conditions

2. Fig. 1
PBC results from a combination of multiple genetic and environmental factors that may affect the immune system and the liver.
Journal of Hepatology  , DOI: ( /j.jhep )
Copyright © 2010 European Association for the Study of the Liver Terms and Conditions

3. Fig. 2
PBC sequential events: from PDCE2-breakdown tolerance to cirrhosis and liver failure.
Journal of Hepatology  , DOI: ( /j.jhep )
Copyright © 2010 European Association for the Study of the Liver Terms and Conditions

4. Fig. 3
(A) Typical aspect of the lymphocytic interface hepatitis in PBC; (B) biliary piecemeal necrosis with marked increase of ductular profiles accompanied by oedema, neutrophils, and fibroplasia.
Journal of Hepatology  , DOI: ( /j.jhep )
Copyright © 2010 European Association for the Study of the Liver Terms and Conditions

5. Fig. 4
Schematic representation of the putative sequence events in the fibrogenesis process of PBC (ET1: endothelin 1, PDGF: platelet-derived growth factor, VEGF: vascular epithelial growth factor, CCL2: chemokine ligand 2, TGFβ: transforming growth factor β, NGF: nerve growth factor, BMD: bone marrow derived-cells, EMT: epithelial–mesenchymal transition).
Journal of Hepatology  , DOI: ( /j.jhep )
Copyright © 2010 European Association for the Study of the Liver Terms and Conditions

6. Fig. 5
Patterns of development of ductopenia and fibrosis in the three major clinical forms of PBC.
Journal of Hepatology  , DOI: ( /j.jhep )
Copyright © 2010 European Association for the Study of the Liver Terms and Conditions