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Published byEgbert McCormick Modified over 6 years ago
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Searching for a common mechanism for placenta-mediated pregnancy complications and cardiovascular disease: role of lipoprotein(a) Ilaria Romagnuolo, M.Sc., Elena Sticchi, M.Sc., Ph.D., Monica Attanasio, M.Sc., Elisa Grifoni, M.D., Gabriele Cioni, M.D., Ph.D., Anna Paola Cellai, M.Sc., Rosanna Abbate, M.D., Cinzia Fatini, M.D., Ph.D. Fertility and Sterility Volume 105, Issue 5, Pages e3 (May 2016) DOI: /j.fertnstert Copyright © 2016 American Society for Reproductive Medicine Terms and Conditions
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Figure 1 Lipoprotein (a) [Lp(a)] levels according to each placenta-mediated pregnancy complications (PMPC). CNTRL = control; PE = preeclampsia; SB = stillbirth; SGA = small-for-gestational-age neonates. Fertility and Sterility , e3DOI: ( /j.fertnstert ) Copyright © 2016 American Society for Reproductive Medicine Terms and Conditions
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Figure 2 Lipoprotein (a) [Lp(a)] lconcentrations according to LPA gene unfavorable alleles burden. Fertility and Sterility , e3DOI: ( /j.fertnstert ) Copyright © 2016 American Society for Reproductive Medicine Terms and Conditions
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Supplemental Figure 1 Flow chart. PE = preeclampsia; SB = stillbirth; SGA = small-for-gestational-age neonates. Fertility and Sterility , e3DOI: ( /j.fertnstert ) Copyright © 2016 American Society for Reproductive Medicine Terms and Conditions
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