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Hyperoxaluria: a gut–kidney axis?
Stef Robijn, Bernd Hoppe, Benjamin A. Vervaet, Patrick C. D'Haese, Anja Verhulst Kidney International Volume 80, Issue 11, Pages (December 2011) DOI: /ki Copyright © 2011 International Society of Nephrology Terms and Conditions
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Figure 1 Overview of endogenous oxalate synthesis pathways. PH I–III, primary hyperoxaluria types I–III. Kidney International , DOI: ( /ki ) Copyright © 2011 International Society of Nephrology Terms and Conditions
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Figure 2 Proposed mechanism of renal oxalate handling. Cl−, chloride; DCT, distal convoluted tubule; OMCD, outer medulla collecting duct; Ox2−, oxalate; PCT, proximal convoluted tubule; PST, proximal straight tubule; SLC26, solute-linked carrier 26; SO42−, sulfate; TAL, thick ascending limb. Kidney International , DOI: ( /ki ) Copyright © 2011 International Society of Nephrology Terms and Conditions
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Figure 3 Proposed mechanism of oxalate handling across liver, stomach, and intestinal tract. Cl−, chloride; Ox2−, oxalate; SLC26, solute-linked carrier 26; SO42−, sulfate. Kidney International , DOI: ( /ki ) Copyright © 2011 International Society of Nephrology Terms and Conditions
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