1. Pulmonary Hypertension after Hematopoietic Stem Cell Transplantation
Christopher E. Dandoy, Russel Hirsch, Ranjit Chima, Stella M. Davies, Sonata Jodele 
Biology of Blood and Marrow Transplantation 
Volume 19, Issue 11, Pages (November 2013)
DOI: /j.bbmt
Copyright © 2013 American Society for Blood and Marrow Transplantation Terms and Conditions

2. Figure 1
Lung pathology of pulmonary hypertension. (A) Lung pathology of pulmonary arterial hypertension. Severe muscle proliferation (a) and intimal proliferation (b), with almost complete obliteration of the vessel lumen (c) in a patient with severe PH. The internal (d) and external elastic lamina (e) are still present. (B) Plexogenic pulmonary arteriopathy. Complicated plexogenic lesions in a completely obliterated vessel in a patient with severe PH. Multiple tortuous vessels are seen in the previous vessel lumen (arrows). (C and D) PVOD; patchy thickening of alveolar septa and intimal fibrosis narrowing the pulmonary veins (arrows).
(Reprinted from Miura et al. [55] with permission from Elsevier). A, B, and C are H & E–stained images at 40×. D is an elastica-van Gieson–stained image at 40×.
Biology of Blood and Marrow Transplantation  , DOI: ( /j.bbmt )
Copyright © 2013 American Society for Blood and Marrow Transplantation Terms and Conditions

3. Figure 2
Treatment algorithm for PH unresponsive to first-line therapy of hemodynamically compromising PH, reflecting clinical practice. PDE-5, phosphodiesterase-5; ERA, endothelin receptor antagonist.
(Algorithm adopted from [29,32].)
Biology of Blood and Marrow Transplantation  , DOI: ( /j.bbmt )
Copyright © 2013 American Society for Blood and Marrow Transplantation Terms and Conditions

4. Figure 3 Proposed algorithm for evaluation of possible PH after HSCT.
(Algorithm adopted from Jodele et al. [22].)
Biology of Blood and Marrow Transplantation  , DOI: ( /j.bbmt )
Copyright © 2013 American Society for Blood and Marrow Transplantation Terms and Conditions