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Figure 3 Examples of gene expression heterogeneity

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Presentation on theme: "Figure 3 Examples of gene expression heterogeneity"— Presentation transcript:

1 Figure 3 Examples of gene expression heterogeneity
in epigenome regulators among the TCGA gastric cancer subtypes Figure 3 | Examples of gene expression heterogeneity in epigenome regulators among the TCGA gastric cancer subtypes. a | Numbers indicate average reads per kilobase per million mapped reads (RPKM) values. Colour code represents relative transcript levels (red, high; green, low). Epstein–Barr virus (EBV), n = 25; microsatellite instability (MSI), n = 64; chromosomal instability (CIN), n = 142; genomically stable (GS), n = 60. b | TET1 mRNA levels in the four TCGA subtypes. This figure was generated using publicly accessible data from The Cancer Genome Atlas147. Padmanabhan, N. et al. (2017) How to stomach an epigenetic insult: the gastric cancer epigenome Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro

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2017.01.04 최장현.

최장현.
Figure 1 Number of somatic mutation rates across The Cancer Genome Atlas (TCGA) projects Figure 1 | Number of somatic mutation rates across The Cancer.

Figure 1 Number of somatic mutation rates across The Cancer Genome Atlas (TCGA) projects Figure 1 | Number of somatic mutation rates across The Cancer.
Figure 2 Enteroids can model transport physiology

Figure 2 Enteroids can model transport physiology
promotes the oncogenic activity of CagA

promotes the oncogenic activity of CagA
Fig. 8. Recurrent copy number amplification of BRD4 gene was observed across common cancers. Recurrent copy number amplification of BRD4 gene was observed.

Fig. 8. Recurrent copy number amplification of BRD4 gene was observed across common cancers. Recurrent copy number amplification of BRD4 gene was observed.
Figure 4 Activation of clopidogrel via cytochrome P450

Figure 4 Activation of clopidogrel via cytochrome P450
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro

Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Patients cured of HCV infection

Figure 1 Patients cured of HCV infection
Nat. Rev. Urol. doi: /nrurol

Nat. Rev. Urol. doi: /nrurol
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Figure 1 Worldwide incidence of CCA
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro

Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro

Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 1 Biosimilar development process

Figure 1 Biosimilar development process
Figure 2 Effect of PPIs on gastric physiology

Figure 2 Effect of PPIs on gastric physiology
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro

Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 4 Giant lipid droplet formation

Figure 4 Giant lipid droplet formation
Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro

Nat. Rev. Gastroenterol. Hepatol. doi: /nrgastro
Figure 2 The association between CD8+ T‑cell density of the tumour

Figure 2 The association between CD8+ T‑cell density of the tumour
Figure 6 Combination therapy for HCC

Figure 6 Combination therapy for HCC
Figure 2 Modelling the effect of HCV treatment on reinfection in people who inject drugs Figure 2 | Modelling the effect of HCV treatment on reinfection.

Figure 2 Modelling the effect of HCV treatment on reinfection in people who inject drugs Figure 2 | Modelling the effect of HCV treatment on reinfection.

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