1. Fig Reduced beta cell mass in type 2 diabetic patients, as compared to non-diabetic controls, although there is a marked inter-subject variability and clear overlap between the two groups (adapted from [21])
The Islets of Langerhans, Islam, Md. Shahidul (Ed.)
ISBN: , Springer

2. Fig Isolated type 2 diabetic (T2DM) islets show Increased apoptosis and enhanced caspase-3 and caspase-8 activities in isolated type 2 diabetic (T2DM) islets, as compared to non-diabetic controls. Adapted from [22]
The Islets of Langerhans, Islam, Md. Shahidul (Ed.)
ISBN: , Springer

3. Fig ATP production and ATP/ADP ratio increase in non-diabetic but not in type 2 diabetic islets following exposure to 3.3–16.7 mmol/l glucose concentration. Adapted from [27]
The Islets of Langerhans, Islam, Md. Shahidul (Ed.)
ISBN: , Springer

4. Fig A significant induction of genes involved in endoplasmic reticulum stress (BiP and XBP-1t) in isolated type 2 diabetic islets at increased glucose concentration. Adapted from [23]
The Islets of Langerhans, Islam, Md. Shahidul (Ed.)
ISBN: , Springer

5. Fig Figure demonstrating the increased the amount of insulin granules in type 2 diabetic beta cells that are pre-exposed to metformin
(reproduced with modifications from [22])
The Islets of Langerhans, Islam, Md. Shahidul (Ed.)
ISBN: , Springer