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Adrenocortical hyperresponsivity to adrenocorticotropic hormone: a mechanism favoring the normal production of cortisol in 21-hydroxylase-deficient nonclassic.

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Presentation on theme: "Adrenocortical hyperresponsivity to adrenocorticotropic hormone: a mechanism favoring the normal production of cortisol in 21-hydroxylase-deficient nonclassic."— Presentation transcript:

1 Adrenocortical hyperresponsivity to adrenocorticotropic hormone: a mechanism favoring the normal production of cortisol in 21-hydroxylase-deficient nonclassic adrenal hyperplasia  Raquel Huerta, M.D., Didier Dewailly, M.D., Christine Decanter, M.D., Eric S Knochenhauer, M.D., Larry R Boots, M.D., Ricardo Azziz, M.D.  Fertility and Sterility  Volume 74, Issue 2, Pages (August 2000) DOI: /S (00)

2 Figure 1 Basal (Steroid0) and ACTH-(1-24)-stimulated (Steroid60) levels of 11-deoxycortisol (S) and cortisol (F) in 24 patients with 21-hydroxylase deficient nonclassic adrenal hyperplasia (NCAH) and 37 controls. Depicted are the median values, and brackets represent the range. A statistically significant difference in the basal and stimulated levels between NCAH and controls is observed for S, but not F. Huerta. Adrenocortical hyperresponsivity. Fertil Steril 2000. Fertility and Sterility  , DOI: ( /S (00) )

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