1. Hypermethylation of estrogen receptor-α gene in atheromatosis patients and its correlation with homocysteine 
Yu-Shan Huang, Yan-Fang Zhi, Shu-Ren Wang 
Pathophysiology 
Volume 16, Issue 4, Pages (October 2009)
DOI: /j.pathophys
Copyright © 2009 Elsevier Ireland Ltd Terms and Conditions

2. Fig. 1
Electrophoregram of methylation status with different methylation level in ER-α gene (I) fully methylation sample, (II) half-methylation sample, and (III) non-methylation sample. U: indicated the unmethylated PCR band with primers ER-pU matching the sequences that the cytosine in giving DNA sequences were unmethylated, which would be converted to thymidine after sodium bisulfite treatment. M: indicated the methylated PCR band with primers ER-pM matching the sequences that the cytosine in given DNA sequences were methylated, which would not be influenced by sodium bisulfite treatment, remained to be cytosine.
Pathophysiology  , DOI: ( /j.pathophys )
Copyright © 2009 Elsevier Ireland Ltd Terms and Conditions

3. Fig. 2
The alteration of methylation status of ERα gene treated by Hcy with different concentrations and different treating time. (U and M: see Fig. 1 legend).
Pathophysiology  , DOI: ( /j.pathophys )
Copyright © 2009 Elsevier Ireland Ltd Terms and Conditions

4. Fig. 3
Semiquantitative RT-PCR assessment of ERα gene expression in HUSMCs treated by Hcy with different treating time **p<0.01 vs control (no drug group).
Pathophysiology  , DOI: ( /j.pathophys )
Copyright © 2009 Elsevier Ireland Ltd Terms and Conditions

5. Fig. 4
Effect of Hcy on cell viability: dose-dependent promoting effect of Hcy concentration on SMC proliferation.
Pathophysiology  , DOI: ( /j.pathophys )
Copyright © 2009 Elsevier Ireland Ltd Terms and Conditions