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Published byCarlos Eduardo Cipriano Modified over 5 years ago
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IgA Autoimmune Disorders: Development of a Passive Transfer Mouse Model
John J. Zone, C. Anthony Egan, Ted B. Taylor Journal of Investigative Dermatology Symposium Proceedings Volume 9, Issue 1, Pages (January 2004) DOI: /j x Copyright © 2004 The Society for Investigative Dermatology, Inc Terms and Conditions
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Figure 1 Diagrammatic representation of the transmembrane hemidesmosomal protein BP180 (BPAg2). The primary site of antigenic reactivity for bullous pemphigoid (MCW1) is different from that for linear IgA bullous dermatosis (LABD97). Journal of Investigative Dermatology Symposium Proceedings 2004 9, 47-51DOI: ( /j x) Copyright © 2004 The Society for Investigative Dermatology, Inc Terms and Conditions
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Figure 2 Infiltration of dermal papillae with neutrophils and mononuclear cells after administration of IgA BMZ antibodies (H&E 40 X). Journal of Investigative Dermatology Symposium Proceedings 2004 9, 47-51DOI: ( /j x) Copyright © 2004 The Society for Investigative Dermatology, Inc Terms and Conditions
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Figure 3 BMZ vesicle with neutrophil infiltration after administration of IgA BMZ antibodies (H&E 40X). Journal of Investigative Dermatology Symposium Proceedings 2004 9, 47-51DOI: ( /j x) Copyright © 2004 The Society for Investigative Dermatology, Inc Terms and Conditions
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