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Cerebral angioendotheliomatosis associated with hemispheric symptoms of carotid arterial origin
James E. Chapman, M.D., William Allen Loy, M.D. Journal of Vascular Surgery Volume 2, Issue 2, Pages (March 1985) DOI: / (85) Copyright © 1985 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions
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Fig. 1 Malignant cells proliferating in lumen and infiltrating wall of blood vessel of cerebral cortex. Lymphocytes surround vessel and adjacent cerebral cortex shows degenerative changes. (Hematoxylin-cosin stain; original magnification × 100.) Journal of Vascular Surgery 1985 2, DOI: ( / (85) ) Copyright © 1985 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions
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Fig. 2 Fibrinoid necrosis of wall of cerebellar artery with malignant cells filling lumen. Shrinkage and loss of Purkinje cells secondary to hypoxia. (Hematoxylin-eosin stain; original magnification × 40.) Journal of Vascular Surgery 1985 2, DOI: ( / (85) ) Copyright © 1985 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions
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Fig. 3 Leptomeningeal blood vessel filled with malignant cells, which also infiltrate wall. (Hematoxylin-eosin stain; original magnification × 100.) Journal of Vascular Surgery 1985 2, DOI: ( / (85) ) Copyright © 1985 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions
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Fig. 4 Clusters of malignant cells partially obliterating lumen of periadrenal blood vessels. (Hematoxylin-eosin stain; original magnification × 40.) Journal of Vascular Surgery 1985 2, DOI: ( / (85) ) Copyright © 1985 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions
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