1. Volume 69, Issue 1, Pages 262-264 (July 2018)
Erratum to “A new HDV mouse model identifies mitochondrial antiviral signaling protein (MAVS) as a key player in IFN-β induction” [J Hepatol 67 (2017) 669–679] 
Lester Suárez-Amarán, Carla Usai, Marianna Di Scala, Cristina Godoy, Yi Ni, Mirja Hommel, Laura Palomo, Víctor Segura, Cristina Olagüe, Africa Vales, Alicia Ruiz-Ripa, Maria Buti, Eduardo Salido, Jesús Prieto, Stephan Urban, Francisco Rodríguez-Frias, Rafael Aldabe, Gloria González-Aseguinolaza 
Journal of Hepatology 
Volume 69, Issue 1, Pages (July 2018)
DOI: /j.jhep
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2. Fig. 1
Infection of C57BL/6 mice with AAV-HBV/HDV gives rise to the production of circulating HDV infectious particles and a detectable humoral immune response against viral antigens, while anti-HBsAg and anti-HDAg humoral response is induced. (This figure appears in colour on the web.)
Journal of Hepatology  , DOI: ( /j.jhep )
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3. Fig. 2
Infection of RAGB6 mice with AAV-HBV/HDV gives rise to the sustained production of circulating HDV infectious particles and HBV particles. (This figure appears in colour on the web.)
Journal of Hepatology  , DOI: ( /j.jhep )
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4. Fig. 8
AAV-HDV infection induces an innate immune response that is partially dependent on MAVS. (This figure appears in colour on the web.)
Journal of Hepatology  , DOI: ( /j.jhep )
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