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Coexistent aortic dissection and hepatocellular carcinoma with elevated plasma transforming growth factor β level: Possible roles of fibrillin 1 and transforming.

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Presentation on theme: "Coexistent aortic dissection and hepatocellular carcinoma with elevated plasma transforming growth factor β level: Possible roles of fibrillin 1 and transforming."— Presentation transcript:

1 Coexistent aortic dissection and hepatocellular carcinoma with elevated plasma transforming growth factor β level: Possible roles of fibrillin 1 and transforming growth factor β  Hisato Takagi, MD, PhD, Hideaki Manabe, MD, Seishiro Sekino, MD, Takayoshi Kato, MD, Yukihiro Matsuno, MD, PhD, Takuya Umemoto, MD, PhD  The Journal of Thoracic and Cardiovascular Surgery  Volume 129, Issue 2, Pages (February 2005) DOI: /j.jtcvs Copyright © 2005 The American Association for Thoracic Surgery Terms and Conditions

2 Figure 1 Computed tomographic scanning disclosed both Stanford type A acute aortic dissection (A) and hepatic tumor, relatively hypodense in delayed phase (B). Postoperative computed tomographic scanning revealed hepatic lesion to be hyperdense in arterial phase (C). The Journal of Thoracic and Cardiovascular Surgery  , DOI: ( /j.jtcvs ) Copyright © 2005 The American Association for Thoracic Surgery Terms and Conditions


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