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Cerebral Hyperperfusion Syndrome After Surgical Repair of Congenital Supravalvular Aortic Stenosis
Ting-Wei Lin, MD, MS, Jieh-Neng Wang, MD, Chung-Dann Kan, MD, PhD The Annals of Thoracic Surgery Volume 100, Issue 3, Pages e51-e54 (September 2015) DOI: /j.athoracsur Copyright © 2015 The Society of Thoracic Surgeons Terms and Conditions
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Fig 1 (A) Contrast-enhanced computed tomographic (CT) image showing extreme focal stenosis over the ascending aorta just above the sinotubular junction. The size of the aortic root and the distal ascending aorta are normal. (B) Postoperative CT showing good correction of the stenosis. The Annals of Thoracic Surgery , e51-e54DOI: ( /j.athoracsur ) Copyright © 2015 The Society of Thoracic Surgeons Terms and Conditions
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Fig 2 Computed tomographic image of the brain on postoperative day 2, when the patient had altered consciousness, showing global cerebral edema with sulci effacement. No intracranial lesion was identified, and thus vasogenic cerebral edema was favored. The Annals of Thoracic Surgery , e51-e54DOI: ( /j.athoracsur ) Copyright © 2015 The Society of Thoracic Surgeons Terms and Conditions
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Fig 3 (A) T1-weighted, (B) T2-weighted, (C) diffusion-weighted, and (D) apparent diffusion coefficient magnetic resonance images after medical management of cerebral hyperperfusion syndrome 3 days later, showing much improvement of the cerebral edema with minimal subdural hematoma at the left parietooccipital area, thought to be caused by rapid decline of the intracranial pressure and tearing of the bridging vein after aggressive management of cerebral edema. The Annals of Thoracic Surgery , e51-e54DOI: ( /j.athoracsur ) Copyright © 2015 The Society of Thoracic Surgeons Terms and Conditions
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