Presentation is loading. Please wait.

Presentation is loading. Please wait.

Transcriptomic and epigenetic changes associated with Factor 1 in the scMT data Transcriptomic and epigenetic changes associated with Factor 1 in the scMT.

Published byDjaja Kusuma Modified over 6 years ago

Similar presentations

Presentation on theme: "Transcriptomic and epigenetic changes associated with Factor 1 in the scMT data Transcriptomic and epigenetic changes associated with Factor 1 in the scMT."— Presentation transcript:

1 Transcriptomic and epigenetic changes associated with Factor 1 in the scMT data
Transcriptomic and epigenetic changes associated with Factor 1 in the scMT data RNA expression changes for the top 20 genes with largest weight on Factor 1.DNA methylation rate changes for the top 20 CpG sites with largest weight. Shown is a non‐linear loess regression model fit per CpG site.RNA expression changes for the top 20 genes with largest weight on Factor 2. Ricard Argelaguet et al. Mol Syst Biol 2018;14:e8124 © as stated in the article, figure or figure legend

Download ppt "Transcriptomic and epigenetic changes associated with Factor 1 in the scMT data Transcriptomic and epigenetic changes associated with Factor 1 in the scMT."

Similar presentations

GFP‐Sed5p localization defect in sgt2Δ.

GFP‐Sed5p localization defect in sgt2Δ.
Statistical analysis of transgene expression in pigs generated by lentiviral gene transfer. Statistical analysis of transgene expression in pigs generated.

Statistical analysis of transgene expression in pigs generated by lentiviral gene transfer. Statistical analysis of transgene expression in pigs generated.
Statistical colorings of 1H NMR estimates of biochemical variables.

Statistical colorings of 1H NMR estimates of biochemical variables.
DNase‐HS sites are main independent determinants of DNA replication timing Simulations based on genome sequence features (GC content, CpG islands), or.

DNase‐HS sites are main independent determinants of DNA replication timing Simulations based on genome sequence features (GC content, CpG islands), or.
Abundance of proteins matching selected subcellular locations and functions in CaCo‐2 cells. Abundance of proteins matching selected subcellular locations.

Abundance of proteins matching selected subcellular locations and functions in CaCo‐2 cells. Abundance of proteins matching selected subcellular locations.
Schematic diagram of the three circuits analysed in this work.

Schematic diagram of the three circuits analysed in this work.
Network topology reflects target gene expression profile and function.

Network topology reflects target gene expression profile and function.
MRNA coexpression of neighbouring genes is driven by chromatin fluctuations and regulatory interference mRNA coexpression of neighbouring genes is driven.

MRNA coexpression of neighbouring genes is driven by chromatin fluctuations and regulatory interference mRNA coexpression of neighbouring genes is driven.
Summary of all the reactions considered in the SmartCell simulation of the three circuits. Summary of all the reactions considered in the SmartCell simulation.

Summary of all the reactions considered in the SmartCell simulation of the three circuits. Summary of all the reactions considered in the SmartCell simulation.
Analysis of in silico flux changes along the exponential growth phase: (A) In silico flux changes from 24 to 36 h, from 36 to 48 h, and from 48 to 60 h.

Analysis of in silico flux changes along the exponential growth phase: (A) In silico flux changes from 24 to 36 h, from 36 to 48 h, and from 48 to 60 h.
Motif detectability corresponds to the phylogenetic profile of the cognate transcription factor. Motif detectability corresponds to the phylogenetic profile.

Motif detectability corresponds to the phylogenetic profile of the cognate transcription factor. Motif detectability corresponds to the phylogenetic profile.
Characterization of the inferred factor associated with the differentiation state of the cell of origin Characterization of the inferred factor associated.

Characterization of the inferred factor associated with the differentiation state of the cell of origin Characterization of the inferred factor associated.
Epistatic interactions and gene expression variation.

Epistatic interactions and gene expression variation.
Tight regulation of gene expression variation is coupled to promoter architecture. Tight regulation of gene expression variation is coupled to promoter.

Tight regulation of gene expression variation is coupled to promoter architecture. Tight regulation of gene expression variation is coupled to promoter.
Heatmaps of the gene mutation distributions in oncogene‐signaling blocks for six representative cancer types. Heatmaps of the gene mutation distributions.

Heatmaps of the gene mutation distributions in oncogene‐signaling blocks for six representative cancer types. Heatmaps of the gene mutation distributions.
(A) Protein biosynthesis on a chip and in solution.

(A) Protein biosynthesis on a chip and in solution.
Functional analysis of duplicate pair CIK1–VIK1 (A) Genetic interaction profile similarity. Functional analysis of duplicate pair CIK1–VIK1 (A) Genetic.

Functional analysis of duplicate pair CIK1–VIK1 (A) Genetic interaction profile similarity. Functional analysis of duplicate pair CIK1–VIK1 (A) Genetic.
Identification of progenitor states in human cord blood hematopoiesis

Identification of progenitor states in human cord blood hematopoiesis
Relationship between clinical data and latent factors

Relationship between clinical data and latent factors
Structure of in‐frame deletions.

Structure of in‐frame deletions.

Similar presentations

Ads by Google