1. Volume 94, Issue 6, Pages 1035-1037 (December 2018)
Journal Club 
Kidney International 
Volume 94, Issue 6, Pages (December 2018)
DOI: /j.kint
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2. Figure 1
Schematic model of the relationship between Ca2+/CaMK4 signaling and actin stability in podocytes. CAMK, Ca2+/calmodulin-dependent protein kinase; LPS, lipopolysaccharide; P, phosphate; Rac1, Ras-related C3 botulinum toxin substrate 1; TLR, toll-like receptor; TRPC5, transient receptor potential channel 5.
Reprinted with permission from Maeda K, Otomo K, Yoshida N, et al. CaMK4 compromises podocyte function in autoimmune and nonautoimmune kidney disease. J Clin Invest. 2018;128:3445–3459; Copyright © 2018 American Society for Clinical Investigation.
Kidney International  , DOI: ( /j.kint )
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