1. Volume 156, Issue 1, Pages 46-58.e7 (January 2019)
Rome Foundation Working Team Report on Post-Infection Irritable Bowel Syndrome 
Giovanni Barbara, Madhusudan Grover, Premysl Bercik, Maura Corsetti, Uday C. Ghoshal, Lena Ohman, Mirjana Rajilić-Stojanović 
Gastroenterology 
Volume 156, Issue 1, Pages e7 (January 2019)
DOI: /j.gastro
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2. Figure 1
Rome IV diagnostic algorithm for postinfection irritable bowel syndrome.
Gastroenterology  , e7DOI: ( /j.gastro )
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3. Figure 2
Geographic distribution of postinfection irritable bowel syndrome prevalence by pathogen type.
Gastroenterology  , e7DOI: ( /j.gastro )
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4. Figure 3
Schematic representation of putative pathophysiology underlying postinfection irritable bowel syndrome.
Gastroenterology  , e7DOI: ( /j.gastro )
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5. Figure 4
Putative role of infection-associated shifts in microbial community in postinfection IBS. A host with a Clostridiales-predominant microbiota type is likely to remain in the eubiosis state after infection, whereas, presence of a Bacteroidetes-predominant community may predispose to development of long-term dysbiosis upon infection. Dysbiosis can then result in shifts in bile acid composition and the cytokine and immune milieu, which can affect epithelial and neuromuscular function and further perpetuate dysbiosis.
Gastroenterology  , e7DOI: ( /j.gastro )
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6. Figure 5
Summary of animal models for postinfection irritable bowel syndrome. Various pathogen types have unique or overlapping mechanisms associated with irritable bowel syndrome. The proximity to the center reflects the strength of association.
Gastroenterology  , e7DOI: ( /j.gastro )
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7. Figure 6
An expert consensus–based proposed treatment algorithm for PI-IBS.
Gastroenterology  , e7DOI: ( /j.gastro )
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8. Supplementary Figure 1
Postinfection irritable bowel syndrome prevalence with pathogen type and relative risk compared with unexposed cohort in studies examining within 12 months and beyond 12 months after the episode of infectious enteritis.
Gastroenterology  , e7DOI: ( /j.gastro )
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