1. Mechanisms of myocardial protection produced by chronic ethanol consumption 
Paul S. Pagel, Judy R. Kersten, David C. Warltier 
Pathophysiology 
Volume 10, Issue 2, Pages (April 2004)
DOI: /j.pathophys

2. Fig. 1
Myocardial infarct size expressed as a percentage of the area of the left ventricle at risk. CON, control; ETOH, ethanol; WK, week; GLB, glyburide; 5-HD, 5-hydroxydecanoate; HMR, HMR-1098; DPCPX, 8-cyclopentyl-1,3,dipropylxanthine; (*) significantly (P<0.05) different from CON.
Pathophysiology  , DOI: ( /j.pathophys )

3. Fig. 2
Relationship between myocardial infarct size (expressed as a percentage of the left ventricular area at risk) and transmural coronary collateral blood flow in dogs pretreated with water–dog chow (CON; solid circles) or ethanol–dog chow for 12 weeks (12 WK-ETOH; solid triangles). A significant (P<0.05) downward shift of the relationship between infarct size and collateral blood flow was observed in dogs receiving the ethanol–dog chow mixture as compared to those who did not.
Pathophysiology  , DOI: ( /j.pathophys )