1. Nitric oxide in acute renal failure: NOS versus NOS
Michael S. Goligorsky, Sergey V. Brodsky, Eisei Noiri 
Kidney International 
Volume 61, Issue 3, Pages (March 2002)
DOI: /j x
Copyright © 2002 International Society of Nephrology Terms and Conditions

2. Figure 1
Cellular effects of nitric oxide (NO) depend on its concentration, site of release and duration of action. Abbreviations are: NOS, nitric oxide synthase; ROI, reactive oxygen intermediates; GC, guanylate cyclase.
Kidney International  , DOI: ( /j x)
Copyright © 2002 International Society of Nephrology Terms and Conditions

3. Figure 2
Evolution of our understanding of the involvement of NO and sites of its generation on renal injury. (A) Effects limited to the isolated epithelial cells. (B) The role of peroxynitrite and its potential sources in epithelial cell injury. (C) Involvement of eNOS and NO bioavailability in the subsequent development of injury to the proximal epithelial cells. Abbreviations are: NO, nitric oxide, iNOS, inducible nitric oxide synthase; eNOS, endothelial nitric oxide synthase; L-NAME, N-nitro-l-arginine methyl ester; AS-ODN, antisense oligodeoxynucleotide; MPO, myeloperoxidase.
Kidney International  , DOI: ( /j x)
Copyright © 2002 International Society of Nephrology Terms and Conditions

4. Figure 3
Schematic representation of endothelial cell transplantation into the ischemic kidney (left) resulting in the improved renal function (right).
Kidney International  , DOI: ( /j x)
Copyright © 2002 International Society of Nephrology Terms and Conditions

5. Kidney International 2002 61, 855-861DOI: (10. 1046/j. 1523-1755. 2002
Kidney International  , DOI: ( /j x)
Copyright © 2002 International Society of Nephrology Terms and Conditions