1. The Hippocampus 1 Damage to the hippocampus disrupts our memory.
Left = Verbal
Right = Visual and Locations
The hippocampus is the like the librarian for the library which is our brain.

2. Cerebellum 2 Cerebellum – a neural center in the hindbrain
that processes implicit memories.

3. Long-Term Potentiation (LTP) 3
The current theory of how our long-term memory works.
Memory has a neural basis.
LTP is an increase in a synapse’s firing potential after brief, rapid stimulation.
In other words, if you are trying to remember a phone number, the neurons are firing neurotransmitter through the synapse. The neuron gets used to firing in that pattern and essentially learns to fire in that distinct way. It is a form of rehearsal (but for our neurons).

4. Long term potentiation 3
New synapses formed, communication at existing synapses
Neurotransmitters involved with memory
Glutamate
Acetylcholine
Neural networks – neurons fire in a pattern – form of rehearsal for the neurons
USE IT OR LOSE IT

5. Memory damage Anterograde amnesia: Retrograde amnesia:
Loss of memory of events occurring after the injury
Cannot transfer new memories to STM or LTM
Retrograde amnesia:
Loss of memory of events prior to injury

6. storage
Hippocampus and thalamus important in formation of new memories
Memories stored in many different areas of cortex
No single brain structure/ neurotransmitter exclusively involved in memory formation or storage

7. Stress and Memory 4
Stress can lead to the release of hormones that have been shown to assist in LTM.
Similar to the idea of Flashbulb Memory.

8. Stress Hormones & Memory 5
Heightened emotions (stress-related or otherwise) make for stronger memories. Continued stress may disrupt memory.
OBJECTIVE 12| Discuss some ways stress hormones can affect memory.
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