1. Neuronal nitric oxide synthase inhibition sensitizes the tubuloglomerular feedback mechanism after volume expansion 
Russell Brown, Anna Ollerstam, A. Erik, G. Persson 
Kidney International 
Volume 65, Issue 4, Pages (April 2004)
DOI: /j x
Copyright © 2004 International Society of Nephrology Terms and Conditions

2. Figure 1
Proximal tubular stop-flow pressure at different rates of tubular perfusion. Curves are results of fitting of normalized data from rats in a normovolemic control situation (NC) and after 5% extracellular volume expansion (VEC) (A), and from rats treated with combinations of 5% VE and intratubular infusion of Nω-nitro-L-arginine methyl ester (L-NAME) (VEL-NAME) (B), or intraperitoneal administration of 7-nitro indazole (7-NI) (VE7-NI) (C).
Kidney International  , DOI: ( /j x)
Copyright © 2004 International Society of Nephrology Terms and Conditions

3. Figure 2
Single nephron glomerular filtration rate (SNGFR) measured at loop perfusion rates of 0 and 40 nL/min under normovolemic control conditions (NC) (A), after 5% extracellular volume expansion (VEC) (B), and after treatment with combination of 5% VE and intratubular infusion of Nω-nitro-L-arginine methyl ester (L-NAME) (VEL-NAME) (C), or of 5% VE and intraperitoneal administration of 7-nitro indazole (7-NI) (VE7-NI) (D).*P < 0.05 vs. VEC.
Kidney International  , DOI: ( /j x)
Copyright © 2004 International Society of Nephrology Terms and Conditions