1. Sun Exposure: What Molecular Photodermatology Tells Us About Its Good and Bad Sides 
Jean Krutmann, Akimichi Morita, Jin Ho Chung 
Journal of Investigative Dermatology 
Volume 132, Issue 3, Pages (March 2012)
DOI: /jid
Copyright © 2012 The Society for Investigative Dermatology, Inc Terms and Conditions

2. Figure 1
The arylhydrocarbon receptor in human keratinocytes mediates the DNA damage–independent part of the UVB stress response. AhR, arylhydrocarbon receptor; ARNT, AhR nuclear translocator; COX2, cyclooxygenase-2; CYP1, cytochrome P450 1; ERK, extracellular signal–regulated kinase; FICZ, formylindolo-3,2b-carbazole; Hsp90, heat-shock protein 90; MEK, mitogen-activated protein/extracellular signal–regulated kinase.
Journal of Investigative Dermatology  , DOI: ( /jid )
Copyright © 2012 The Society for Investigative Dermatology, Inc Terms and Conditions

3. Figure 2
Infrared (IR) A radiation–induced retrograde signaling in human skin fibroblasts. ERK, extracellular signal–regulated kinase; MAPK, mitogen-activated protein kinase; MMP-1, matrix metalloproteinase; ROS, reactive oxygen species.
Journal of Investigative Dermatology  , DOI: ( /jid )
Copyright © 2012 The Society for Investigative Dermatology, Inc Terms and Conditions

4. Figure 3
The subcutis is a target tissue for solar radiation. MCP-3, monocyte chemotactic protein-3; PPARγ, peroxisome proliferator–activated receptor-γ.
Journal of Investigative Dermatology  , DOI: ( /jid )
Copyright © 2012 The Society for Investigative Dermatology, Inc Terms and Conditions

5. Figure 4
The mode of action of phototherapy. STAT3, signal transducer and activator of transcription-3; Th17, T helper type 17; TNFα, tumor necrosis factor-α; Treg, regulatory T cell.
Journal of Investigative Dermatology  , DOI: ( /jid )
Copyright © 2012 The Society for Investigative Dermatology, Inc Terms and Conditions