1. Volume 69, Issue 1, Pages 68-72 (January 2006)
Fructose-1,6 diphosphate as a protective agent for experimental ischemic acute renal failure 
N. Antunes, C.A. Martinusso, C.M. Takiya, A.J.R. da Silva, J.F.R. de Ornellas, P.R. Elias, M. Leite, L.R. Cardoso 
Kidney International 
Volume 69, Issue 1, Pages (January 2006)
DOI: /sj.ki
Copyright © 2006 International Society of Nephrology Terms and Conditions

2. Figure 1
The glycolytic pathway. Grey headed arrows indicate possible fates and actions of exogenous FDP in glucose metabolism.
Kidney International  , 68-72DOI: ( /sj.ki )
Copyright © 2006 International Society of Nephrology Terms and Conditions

3. Figure 2
ATP levels in renal slices over 72 h of tissue incubation. There was a significant decrease in the C, S and M groups over 72 h. In the F group, there was no decrease in ATP content in the first 24 h, followed by a progressive decrease but levels were kept higher in the F group in comparison to the C, S and M groups at 24, 48 and 72 h.
Kidney International  , 68-72DOI: ( /sj.ki )
Copyright © 2006 International Society of Nephrology Terms and Conditions

4. Figure 3
LDH release from renal slices into medium over 72 h of tissue incubation. Data represent mean±s.e.m. There was a significant increase in LDH release in all groups. From 24 to 72 h the release was in the order of (P<0.05) C>S>M>F.
Kidney International  , 68-72DOI: ( /sj.ki )
Copyright © 2006 International Society of Nephrology Terms and Conditions

5. Figure 4
F-actin concentration in the proximal tubule following 50 min of cold ischemia. Apical F-actin concentration was significantly higher in the F group than in the C, S and M groups.
Kidney International  , 68-72DOI: ( /sj.ki )
Copyright © 2006 International Society of Nephrology Terms and Conditions