Presentation is loading. Please wait.

Presentation is loading. Please wait.

Fig. 4. Expression analysis of Onecut transcription factors during mdDA neuron development.Adjacent coronal sections of E11.5, E12.5 and E13.5 mouse brains.

Published byΑκακιος Φιλιππίδης Modified over 5 years ago

Similar presentations

Presentation on theme: "Fig. 4. Expression analysis of Onecut transcription factors during mdDA neuron development.Adjacent coronal sections of E11.5, E12.5 and E13.5 mouse brains."— Presentation transcript:

1 Fig. 4. Expression analysis of Onecut transcription factors during mdDA neuron development.Adjacent coronal sections of E11.5, E12.5 and E13.5 mouse brains were hybridized with Onecut (Oc)1 (upper panel), Oc2 (medial panel) and Oc3 (lower panel) RNA probes. Expression analysis of Onecut transcription factors during mdDA neuron development.Adjacent coronal sections of E11.5, E12.5 and E13.5 mouse brains were hybridized with Onecut (Oc)1 (upper panel), Oc2 (medial panel) and Oc3 (lower panel) RNA probes. The region shown starts rostrally at the diencephalon and ends caudally at the midbrain. To identify the region, Th in situ hybridization was performed on adjacent sections. Koushik Chakrabarty et al. Biology Open 2012;1: © Published by The Company of Biologists Ltd

Download ppt "Fig. 4. Expression analysis of Onecut transcription factors during mdDA neuron development.Adjacent coronal sections of E11.5, E12.5 and E13.5 mouse brains."

Similar presentations

Fig. 1. In situ hybridization analysis for detecting endogenous mBest1 transcript in whole-brain region. In situ hybridization results from coronal (top.

Fig. 1. In situ hybridization analysis for detecting endogenous mBest1 transcript in whole-brain region. In situ hybridization results from coronal (top.
Structure of abbreviated dystrophins.

Structure of abbreviated dystrophins.
Volume 6, Issue 8, Pages (August 1996)

Volume 6, Issue 8, Pages (August 1996)
In situ confirmations of Sepw1-enriched genes.

In situ confirmations of Sepw1-enriched genes.
Co‐expression of Sorcs1 and Sorcs3 in the hypothalamus

Co‐expression of Sorcs1 and Sorcs3 in the hypothalamus
Fig. 5. Prip silencing enhances the co-localization of GABARAP with insulin vesicles and β-tubulin.Co-localization of GABARAP (green) with insulin (red)

Fig. 5. Prip silencing enhances the co-localization of GABARAP with insulin vesicles and β-tubulin.Co-localization of GABARAP (green) with insulin (red)
Fig. 1. Overview of the nervous system of the adult S. roscoffensis.

Fig. 1. Overview of the nervous system of the adult S. roscoffensis.
Fig. 3. Effect of NH4Cl (0 or 30 mM) on percentage of motile spermatozoa and VAP after 1 and 5 min after activation. Effect of NH4Cl (0 or 30 mM) on percentage.

Fig. 3. Effect of NH4Cl (0 or 30 mM) on percentage of motile spermatozoa and VAP after 1 and 5 min after activation. Effect of NH4Cl (0 or 30 mM) on percentage.
Fig. 2. Outline of the two types of stimulus sequences employed in the analysis.(A) Environment information stimuli; (B) adaptation stimuli. Outline of.

Fig. 2. Outline of the two types of stimulus sequences employed in the analysis.(A) Environment information stimuli; (B) adaptation stimuli. Outline of.
Fig. 3. No increase in Tau-mediated toxicity caused by the R406W mutation.(A) GMR-gal4 driving expression from the 68A integration site of 2N4R or 0N4R.

Fig. 3. No increase in Tau-mediated toxicity caused by the R406W mutation.(A) GMR-gal4 driving expression from the 68A integration site of 2N4R or 0N4R.
Fig. 4. smc3 regulates the expression of cx43 in regenerating fins.

Fig. 4. smc3 regulates the expression of cx43 in regenerating fins.
Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.

Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.
Fig. 1. Muscle-specific PITX1 over-expression in the Pitx1 transgenic mice.(A) Detection of Pitx1 mRNA expression in muscles of the Pitx1 transgenic mice.

Fig. 1. Muscle-specific PITX1 over-expression in the Pitx1 transgenic mice.(A) Detection of Pitx1 mRNA expression in muscles of the Pitx1 transgenic mice.
Fig. 3. Knockdown of cited3 results in increased cell death but it does not affect proliferation.Embryos that are injected with the control MO (A–C), cited3.

Fig. 3. Knockdown of cited3 results in increased cell death but it does not affect proliferation.Embryos that are injected with the control MO (A–C), cited3.
Fig. 6. Cross-section of the stomach wall and spiral intestine of the embryo, stained with PAS. (A) Surface of the stomach wall (SW) and ingested material.

Fig. 6. Cross-section of the stomach wall and spiral intestine of the embryo, stained with PAS. (A) Surface of the stomach wall (SW) and ingested material.
Fig. 6. The cholinergic receptor subunits a6 (Chrna6) and b3 (Chrnb3) are (subset) specifically expressed in mdDA neurons during development.(A) The Chrna6.

Fig. 6. The cholinergic receptor subunits a6 (Chrna6) and b3 (Chrnb3) are (subset) specifically expressed in mdDA neurons during development.(A) The Chrna6.
Fig. 2. Mapping of the interaction domain on coilin for association with the dyskerin complex. Mapping of the interaction domain on coilin for association.

Fig. 2. Mapping of the interaction domain on coilin for association with the dyskerin complex. Mapping of the interaction domain on coilin for association.
Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region.

Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region.
Fig. 4. Increased adipocyte differentiation in Cbx7-KO ES cells

Fig. 4. Increased adipocyte differentiation in Cbx7-KO ES cells
Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.

Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.

Similar presentations

Ads by Google