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Fig. 5. Upregulation of N. vectensis thrombospondin 168100 during regeneration.(A,B) Juvenile polyps were fixed 48 hours following transection and processed.

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Presentation on theme: "Fig. 5. Upregulation of N. vectensis thrombospondin 168100 during regeneration.(A,B) Juvenile polyps were fixed 48 hours following transection and processed."— Presentation transcript:

1 Fig. 5. Upregulation of N. vectensis thrombospondin during regeneration.(A,B) Juvenile polyps were fixed 48 hours following transection and processed for whole mount in situ hybridization with an antisense (A) or sense (B) probe to Nv Upregulation of N. vectensis thrombospondin during regeneration.(A,B) Juvenile polyps were fixed 48 hours following transection and processed for whole mount in situ hybridization with an antisense (A) or sense (B) probe to Nv There is a superficial signal in the column wall of the polyp that appears stronger near the site of transection (arrows). Scale bars = 500 µm. (C) Quantitative PCR reveals a 41.8±11.1 (n = 3) fold increase in Nv transcripts at 48 hours after transection. The fold increase was normalized to the levels of expression of an rRNA control transcript. (D,E) Longitudinal sections through the body wall of an adult N. vectensis polyp at 48 hours after transection were immunostained with anti-NvTSP The antibody labels the glycocalyx lining the epidermis (arrows) in addition to the neuron-like elements in the retractor muscles (rm). The immunostaining of the glycocalyx ends near the oral end of the column (E). gv, gastrovascular cavity; pm, parietal muscle. Scale bars = 25 µm (D,E). Richard P. Tucker et al. Biology Open 2013;2: © Published by The Company of Biologists Ltd

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Volume 125, Issue 6, Pages (December 2003)

Volume 125, Issue 6, Pages (December 2003)
Fig. 6. Ultrastructural changes in the glycocalyx of N

Fig. 6. Ultrastructural changes in the glycocalyx of N
Fig. 8. CCA and ChQ treatment induce accumulation of F-actin rings.

Fig. 8. CCA and ChQ treatment induce accumulation of F-actin rings.
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Fig. 3. Hts and Dlg are in a complex at the postsynaptic membrane of larval NMJs.(A–B″) PLA with HtsM and Dlg antibodies (green) performed on third instar.
Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.

Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.
Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.

Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.
Fig. 6. Hts regulates par-1 and camkII mRNA distribution and levels in the muscle.(A–J) Views of muscles 6/7 in abdominal segment 4, probed for either.

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Fig. 2. abu/pqn genes are expressed in the pharyngeal cuticle

Fig. 2. abu/pqn genes are expressed in the pharyngeal cuticle
Fig. 3. Knockdown of cited3 results in increased cell death but it does not affect proliferation.Embryos that are injected with the control MO (A–C), cited3.

Fig. 3. Knockdown of cited3 results in increased cell death but it does not affect proliferation.Embryos that are injected with the control MO (A–C), cited3.
Fig. 6. Cross-section of the stomach wall and spiral intestine of the embryo, stained with PAS. (A) Surface of the stomach wall (SW) and ingested material.

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OA is found in the central and peripheral nervous systems.

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Fig. 7. Knockdown of Meis1 abolishes CR4. 2-GFP expression

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Directed differentiation of hPSCs to hypothalamic progenitors.

Directed differentiation of hPSCs to hypothalamic progenitors.
Patient and control ONS cells had very large differences in gene expression. Patient and control ONS cells had very large differences in gene expression.

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