1. This Month in Gastroenterology
Jan Tack, John M. Carethers 
Gastroenterology 
Volume 131, Issue 4, Pages (October 2006)
DOI: /j.gastro
Copyright © 2006 American Gastroenterological Association (AGA) Institute Terms and Conditions

2. Figure 1
(Upper panel) Scatter plot of SELDI-TOF MS–based measured intensities and ELISA data for C3a-desArg. The regression analysis revealed good correlation between the SELDI-TOF MS–derived data and quantification of protein concentration with the immunoassay (r = .71). The green squares indicate values from normal individuals, and the red squares those from patients with colorectal cancer. (Lower panel) Summary of all ELISA values for complement C3a-desArg for the healthy individuals (N), sera from patients with colorectal adenomas (P), and with colorectal carcinomas according to UICC stage.
Gastroenterology  , DOI: ( /j.gastro )
Copyright © 2006 American Gastroenterological Association (AGA) Institute Terms and Conditions

3. Figure 2
(A) Probability of remaining free of proven and possible infections in patients receiving ceftriaxone (continuous line) and norfloxacin (dotted line). (B) Probability of remaining free of spontaneous bacterial peritonitis or bacteremia in patients receiving ceftriaxone (continuous line) and norfloxacin (dotted line).
Gastroenterology  , DOI: ( /j.gastro )
Copyright © 2006 American Gastroenterological Association (AGA) Institute Terms and Conditions

4. Figure 3
Kaplan–Meier survival analysis of KRASV12G/Apc+/1638N (RasAPC), KRASV12G, and Apc+/1638N mice. N = 50 mice per genotype. Intergroup difference in survival at P < .001.
Gastroenterology  , DOI: ( /j.gastro )
Copyright © 2006 American Gastroenterological Association (AGA) Institute Terms and Conditions

5. Figure 4
Acetaldehyde (AcCHO) requires c-Abl to inhibit PPAR-γ function through phosphorylation. Isolated human hepatic stellate cells were transduced with adenovirus (pAd) constructs containing β-galactose (β-gal) as a control, wild-type c-Abl (c-Ablwt), and kinase-dead c-Abl (c-AblKD). Cells were then transfected with a vector containing PPAR-γ (pcmx-PPAR-γ) and incubated with [32P]orthophosphate for 3 hours. Cells were then treated with AcCHO for 15 minutes, and assayed for phosphorylated-PPAR-γ (p-PPAR-γ), which has reduced transcriptional activity. Note that AcCHO cannot induce inhibitory phosphorylation of PPAR-γ with the kinase dead form of c-Abl.
Gastroenterology  , DOI: ( /j.gastro )
Copyright © 2006 American Gastroenterological Association (AGA) Institute Terms and Conditions