Presentation is loading. Please wait.

Presentation is loading. Please wait.

SYK activity is required for anti-IgM–induced CD86 expression.

Published byθάνατος Γούσιος Modified over 5 years ago

Similar presentations

Presentation on theme: "SYK activity is required for anti-IgM–induced CD86 expression."— Presentation transcript:

1 SYK activity is required for anti-IgM–induced CD86 expression.
SYK activity is required for anti-IgM–induced CD86 expression. (A and B) An example of splenic B cells from wild-type (A) and Syk-AQL (B) mice treated with or without anti‐IgM F(ab′)2 in the absence or presence of 3-MB-PP1 (10 μM) for 48 h. Cells were examined by flow cytometry for the expression of CD86. (C) Data represent the mean ± SEM for separate analyses of anti-IgM–induced CD86 expression on cells from four wild-type (closed bars) or Syk-AQL (open bars) mice. Means were compared by Student t test. *p < Wen-Horng Wang et al. ImmunoHorizons 2019;3: Copyright © 2019 The Authors

Download ppt "SYK activity is required for anti-IgM–induced CD86 expression."

Similar presentations

Synergistic antitumor effect of anti-CD40/CpG and 14

Synergistic antitumor effect of anti-CD40/CpG and 14
Uptake of T-MPs for DC maturation and antigen presentation.

Uptake of T-MPs for DC maturation and antigen presentation.
Type 17 immunity is increased in STAT1−/− mice.

Type 17 immunity is increased in STAT1−/− mice.
MP cells are generated from naïve cells in the periphery.

MP cells are generated from naïve cells in the periphery.
Volume 6, Issue 6, Pages (March 2014)

Volume 6, Issue 6, Pages (March 2014)
Viral load and CD4+ T cells in CD8+ T cell–depleted YU-2–infected MISTRG mice. Viral load and CD4+ T cells in CD8+ T cell–depleted YU-2–infected MISTRG.

Viral load and CD4+ T cells in CD8+ T cell–depleted YU-2–infected MISTRG mice. Viral load and CD4+ T cells in CD8+ T cell–depleted YU-2–infected MISTRG.
Depo-Provera altered the expression of cell surface markers associated with HIV susceptibility. Depo-Provera altered the expression of cell surface markers.

Depo-Provera altered the expression of cell surface markers associated with HIV susceptibility. Depo-Provera altered the expression of cell surface markers.
Changes in splenic CD4+ and CD8+ T cell subsets during acute and chronic HIV infection in MISTRG mice. Changes in splenic CD4+ and CD8+ T cell subsets.

Changes in splenic CD4+ and CD8+ T cell subsets during acute and chronic HIV infection in MISTRG mice. Changes in splenic CD4+ and CD8+ T cell subsets.
Smaller T cell zone FRC areas in aged spleens.

Smaller T cell zone FRC areas in aged spleens.
VLPs activate DCs and antigen-specific CD8+ T cells via the STING and cGAS pathway. VLPs activate DCs and antigen-specific CD8+ T cells via the STING and.

VLPs activate DCs and antigen-specific CD8+ T cells via the STING and cGAS pathway. VLPs activate DCs and antigen-specific CD8+ T cells via the STING and.
CD8α+ DC-deficient mice are highly susceptible to Lm infection in the absence of CD169+ macrophages. CD8α+ DC-deficient mice are highly susceptible to.

CD8α+ DC-deficient mice are highly susceptible to Lm infection in the absence of CD169+ macrophages. CD8α+ DC-deficient mice are highly susceptible to.
Exogenous IL-4 cannot rescue Th2 cytokine expression in HuR-deficient cells. Exogenous IL-4 cannot rescue Th2 cytokine expression in HuR-deficient cells.

Exogenous IL-4 cannot rescue Th2 cytokine expression in HuR-deficient cells. Exogenous IL-4 cannot rescue Th2 cytokine expression in HuR-deficient cells.
Altered cytokine production by Klrk1−/− NOD CTL

Altered cytokine production by Klrk1−/− NOD CTL
Role of IL-27 in expression of PD-L1, LAG-3, and CTLA-4 depends on strength of α-CD3/28 signaling. Role of IL-27 in expression of PD-L1, LAG-3, and CTLA-4.

Role of IL-27 in expression of PD-L1, LAG-3, and CTLA-4 depends on strength of α-CD3/28 signaling. Role of IL-27 in expression of PD-L1, LAG-3, and CTLA-4.
Viral fusion is required for DC activation.

Viral fusion is required for DC activation.
CD169+ macrophages mediate Lm translocation to the splenic T cell zones. CD169+ macrophages mediate Lm translocation to the splenic T cell zones. (A) Confocal.

CD169+ macrophages mediate Lm translocation to the splenic T cell zones. CD169+ macrophages mediate Lm translocation to the splenic T cell zones. (A) Confocal.
LV activation of DCs and subsequent CD8+ T cell priming are dependent on STING and cGAS but not on MyD88, TRIF, or MAVS. LV activation of DCs and subsequent.

LV activation of DCs and subsequent CD8+ T cell priming are dependent on STING and cGAS but not on MyD88, TRIF, or MAVS. LV activation of DCs and subsequent.
Immune cell populations, activation, and NKG2D and H60a expression in the spleen of untreated and antibiotic-treated Klrk1+/+ and Klrk1−/− NOD mice. Immune.

Immune cell populations, activation, and NKG2D and H60a expression in the spleen of untreated and antibiotic-treated Klrk1+/+ and Klrk1−/− NOD mice. Immune.
Loss of DGKζ and Cbl-b results in a greater percentage of splenic CD8+ T cells with an activated phenotype. Loss of DGKζ and Cbl-b results in a greater.

Loss of DGKζ and Cbl-b results in a greater percentage of splenic CD8+ T cells with an activated phenotype. Loss of DGKζ and Cbl-b results in a greater.
IFN-γ induces TNF family ligand protein expression in vitro and in vivo. IFN-γ induces TNF family ligand protein expression in vitro and in vivo. (A and.

IFN-γ induces TNF family ligand protein expression in vitro and in vivo. IFN-γ induces TNF family ligand protein expression in vitro and in vivo. (A and.

Similar presentations

Ads by Google