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Fig. 3 H3.1-dependent propagation of H3K27me3 maintains the memory of silencing during DNA replication. H3.1-dependent propagation of H3K27me3 maintains.

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Presentation on theme: "Fig. 3 H3.1-dependent propagation of H3K27me3 maintains the memory of silencing during DNA replication. H3.1-dependent propagation of H3K27me3 maintains."— Presentation transcript:

1 Fig. 3 H3.1-dependent propagation of H3K27me3 maintains the memory of silencing during DNA replication. H3.1-dependent propagation of H3K27me3 maintains the memory of silencing during DNA replication. (A) GR-LhG4 expressed under the control of the HTR13 promoter (solid arrow) can be activated by DEX and subsequently binds to the pOp3 promoter to activate amiRHTR13-1 transcription (dashed arrow). (B) Experimental design of treatment with DEX and the cell cycle inhibitors olomoucine (OLO) and roscovitine (ROS). (C) Gene expression upon DEX and cell cycle inhibitor treatment. (D) Relative H3K27me3 levels at AT5G60250 and AT5G60610 chromatin in seedlings treated with DEX and cell cycle inhibitors. For all experiments, error bars represent SD from three biological replicates. Unpaired t test: *P < 0.05; **P < 0.01; ***P < 0.001; n.s., not significant. Danhua Jiang, and Frédéric Berger Science 2017;357: Copyright © 2017 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works

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