Presentation is loading. Please wait.

Presentation is loading. Please wait.

Short-term synaptic depression in SCN neurons during stimulus train application. Short-term synaptic depression in SCN neurons during stimulus train application.

Published byDominique Leduc Modified over 5 years ago

Similar presentations

Presentation on theme: "Short-term synaptic depression in SCN neurons during stimulus train application. Short-term synaptic depression in SCN neurons during stimulus train application."— Presentation transcript:

1 Short-term synaptic depression in SCN neurons during stimulus train application.
Short-term synaptic depression in SCN neurons during stimulus train application. Stimulation of the optic chiasm with trains of 25 stimuli at to 25-Hz frequencies (A–F). The amplitude of each subsequent eEPSC in the train was normalized to the amplitude of the first eEPSC: eEPSCn/eEPSC1. Frequency-dependence changes of eEPSC amplitude in WT (n = 18 neurons), control (Opn4+/+; Vglut2loxP/loxP; n = 16 neurons), cKO (Opn4Cre/+; Vglut2loxP/loxP; n = 8 neurons), and dKO (Opn4Cre/Cre; Vglut2loxP/loxP; n = 2 neurons) mice during stimulation with frequencies ranging from 0.08 to 25 Hz. Michael G. Moldavan et al. eNeuro 2018;5:ENEURO ©2018 by Society for Neuroscience

Download ppt "Short-term synaptic depression in SCN neurons during stimulus train application. Short-term synaptic depression in SCN neurons during stimulus train application."

Similar presentations

Impaired learning and relearning of virtual cued goal localization in Shank2-deficient mice. Impaired learning and relearning of virtual cued goal localization.

Impaired learning and relearning of virtual cued goal localization in Shank2-deficient mice. Impaired learning and relearning of virtual cued goal localization.
Surface renderings of the brain activation showing significant activation in the general mixed design ANOVA for the interaction between all three factors,

Surface renderings of the brain activation showing significant activation in the general mixed design ANOVA for the interaction between all three factors,
A, ACPs establish direct synaptic contact with AOB GCs

A, ACPs establish direct synaptic contact with AOB GCs
Activation of PBN and CeA by illuminating the hindpaw after PNI

Activation of PBN and CeA by illuminating the hindpaw after PNI
Active avoidance is impaired in aged mice in both multimodal and visual versions of the task. Active avoidance is impaired in aged mice in both multimodal.

Active avoidance is impaired in aged mice in both multimodal and visual versions of the task. Active avoidance is impaired in aged mice in both multimodal.
Peptidergic nociceptor markers are reduced in DRG from Nav-Tsc2 mice.

Peptidergic nociceptor markers are reduced in DRG from Nav-Tsc2 mice.
Synaptic background activity.

Synaptic background activity.
Synaptic transmission and plasticity is disturbed in 2-week-old but not in adult SynDIG1 β-gal mutant mice. Synaptic transmission and plasticity is disturbed.

Synaptic transmission and plasticity is disturbed in 2-week-old but not in adult SynDIG1 β-gal mutant mice. Synaptic transmission and plasticity is disturbed.
A–E, Active avoidance (A–D) and optokinetic testing (E) in an independent cohort. A–E, Active avoidance (A–D) and optokinetic testing (E) in an independent.

A–E, Active avoidance (A–D) and optokinetic testing (E) in an independent cohort. A–E, Active avoidance (A–D) and optokinetic testing (E) in an independent.
A, Multivariate glm analysis for the aggregate observer (for the interval range within –450 and 250 ms from action execution). A, Multivariate glm analysis.

A, Multivariate glm analysis for the aggregate observer (for the interval range within –450 and 250 ms from action execution). A, Multivariate glm analysis.
Computer simulation on a single myelinated fiber with the dimensions of the mouse optic nerve. Computer simulation on a single myelinated fiber with the.

Computer simulation on a single myelinated fiber with the dimensions of the mouse optic nerve. Computer simulation on a single myelinated fiber with the.
Sparse excision of PirB at E15

Sparse excision of PirB at E15
Neuronal ATM level is reduced in mouse models of AD

Neuronal ATM level is reduced in mouse models of AD
Neuronal responses to intracortical microstimulation in V1.

Neuronal responses to intracortical microstimulation in V1.
Simultaneously recorded patterns of wheel running (RPM; black solid lines) and core body temperature (red broken lines) in three individual mice housed.

Simultaneously recorded patterns of wheel running (RPM; black solid lines) and core body temperature (red broken lines) in three individual mice housed.
CT and CI odor responses.

CT and CI odor responses.
Loss of αsyn does not affect mitochondrial-derived ATP levels at the nerve terminal. Loss of αsyn does not affect mitochondrial-derived ATP levels at the.

Loss of αsyn does not affect mitochondrial-derived ATP levels at the nerve terminal. Loss of αsyn does not affect mitochondrial-derived ATP levels at the.
Loss of Cav1 function does not disrupt formation of dual projecting CPN/FPN axonal projections at P8. Loss of Cav1 function does not disrupt formation.

Loss of Cav1 function does not disrupt formation of dual projecting CPN/FPN axonal projections at P8. Loss of Cav1 function does not disrupt formation.
Leptin-induced LTP at TA-CA1 synapses involves a postsynaptic expression mechanism. Leptin-induced LTP at TA-CA1 synapses involves a postsynaptic expression.

Leptin-induced LTP at TA-CA1 synapses involves a postsynaptic expression mechanism. Leptin-induced LTP at TA-CA1 synapses involves a postsynaptic expression.
GABAA-TONIC receptor protein-IR did not vary between the dorsal and ventral SCN at any time point in either LD or DD (A–F). GABAA-TONIC receptor protein-IR.

GABAA-TONIC receptor protein-IR did not vary between the dorsal and ventral SCN at any time point in either LD or DD (A–F). GABAA-TONIC receptor protein-IR.

Similar presentations

Ads by Google